Rabbit IgG, polyclonal - Isotype Control (ChIP Grade) (Ab171870)
详细信息
特异性
The sera for this product is un-immunized, naive sera.
背景信息
Rabbit IgG, polyclonal - Isotype Control (ChIP Grade) (ab171870) is a Rabbit Polyclonal antibody and is validated for use in ChIP, Flow Cyt (Intra), WB. Rabbit IgG, polyclonal - Isotype Control (ChIP Grade) (ab171870) has been cited over 286 times in peer reviewed journals and is trusted by the scientific community. Abcams high quality validation processes ensure Rabbit IgG, polyclonal - Isotype Control (ChIP Grade) (ab171870) has high sensitivity and specificity. Rabbit IgG, polyclonal - Isotype Control (ChIP Grade) (ab171870) specifically detects Isotype control and is sold in 100 ug selling sizes. For more information regarding the isotype control selection, please see Your Guide to Selecting an Isotype Control.
文献引用 (441)
The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer.
组蛋白甲基转移酶 KMT2D 是去势抵抗性前列腺癌谱系可塑性和治疗反应的关键介质。
Heritable changes in chromatin contacts associated with transgenerational susceptibility to diet-induced insulin dysregulation and obesity.
与饮食引起的胰岛素失调和肥胖的跨代易感性相关的染色质接触的遗传性变化。
RORγt(+) APCs require a distinct cis-regulatory element to instruct tolerance to dietary antigens.
RORγt(+) APC 需要独特的顺式调控元件来指导对膳食抗原的耐受性。
Bifurcate Regulation of Hematopoietic Homeostasis and Bone Osteogenesis by VHL-HIF2α-Controlled Adipocyte Function.
VHL-HIF2α控制的脂肪细胞功能对造血稳态和骨形成进行分叉调节。
ZDHHC2-Dependent Palmitoylation Dictates Ferroptosis and Castration Sensitivity in Prostate Cancer via Controlling ACSL4 Degradation and Lipid Peroxidation.
ZDHHC2 依赖的棕榈酰化通过控制 ACSL4 降解和脂质过氧化来决定前列腺癌的铁死亡和去势敏感性。
Targeting the SOX9/TIMP1 Axis with iRGD-Conjugated Nanoplatform Enhances Dendritic Cell Function and Photodynamic Immunotherapy in Gastric Cancer.
利用 iRGD 偶联纳米平台靶向 SOX9/TIMP1 轴可增强树突状细胞功能和光动力免疫疗法在胃癌中的疗效。
Podocyte TLR4 deletion alleviates diabetic kidney disease through prohibiting PKCδ/SHP-1-dependent ER stress and relieving podocyte damage and inflammation.
足细胞 TLR4 缺失通过抑制 PKCδ/SHP-1 依赖的内质网应激，减轻足细胞损伤和炎症，从而缓解糖尿病肾病。
Protease-mediated PRC1 dissociation promotes H2AK119ub remodeling during stress responses.
蛋白酶介导的 PRC1 解离促进应激反应期间 H2AK119ub 的重塑。
Histone demethylase KDM4B epigenetically controls NLRP3 expression to enhance inflammatory responses.
组蛋白去甲基化酶 KDM4B 通过表观遗传学控制 NLRP3 的表达，从而增强炎症反应。
Hepatocyte TonEBP promotes metabolic stress-induced hepatic fibroinflammation involving transcriptional activation of ELR⺠CXC chemokines.
肝细胞TonEBP促进代谢应激诱导的肝脏纤维炎症，涉及ELR⺠CXC趋化因子的转录激活。
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