Anti-ALKBH5 antibody [EPR18958] (Ab195377)
ABH5, OFOXD1, ALKBH5, RNA demethylase ALKBH5, Alkylated DNA repair protein alkB homolog 5, Alpha-ketoglutarate-dependent dioxygenase alkB homolog 5
详细信息
背景信息
What is this antibody validated in? Anti-ALKBH5 antibody [EPR18958] (ab195377) is a rabbit recombinant monoclonal antibody and is validated for use in Western Blot (WB), Immunohistochemistry (IHC-P) in Human, Mouse, Rat samples. What is the molecular weight of ALKBH5? Anti-ALKBH5 [EPR18958] (ab195377) specifically detects a band for ALKBH5 (UniProt: Q6P6C2) at a molecular weight of 44kDa. Trusted by the scientific community Anti-ALKBH5 [EPR18958] (ab195377) was first used in a scientific publication in 2018 and has been cited over 60 times in peer-reviewed journals. Specificity confirmed The specificity of Anti-ALKBH5 antibody [EPR18958] (ab195377) has been confirmed by Western blot testing in ALKBH5 Knockout HEK293T cell line, ab266762. Other related products We have a range of other formats of antibody clone [EPR18958] also available for your convenience: ab195377, Carrier free - ab234528, Alexa Fluor® 647 - ab245891 Patented technology Our RabMAb ® technology is a patented hybridoma-based technology for making rabbit monoclonal antibodies. For details on our patents, please refer to RabMAb ® patents. What are the advantages of a recombinant monoclonal antibody? This product is a recombinant monoclonal antibody, which offers several advantages including - High batch-to-batch consistency and reproducibility - Improved sensitivity and specificity - Long-term security of supply - Animal-free batch production For more information, read more on recombinant antibodies. Trial sizes available! Test your antibody or perform pre-screening before committing to a larger quantity. Sold in 20µl. Discover our selection of trial-size antibodies.
文献引用 (117)
Reversible ALKBH5 cytosolic aggregation accelerates cellular senescence.
ALKBH5胞质可逆聚集加速细胞衰老。
ALKBH5 facilitates Srgn stability via m6A demethylation to disrupt blood-testis barrier integrity in the high-fat diet mice.
ALKBH5 通过 m6A 去甲基化促进 Srgn 的稳定性,从而破坏高脂饮食小鼠的血睾屏障完整性。
Integrative multi-omic profiling of the chronically hypoxic heart: focus on m(6)A and m(6)Am epitranscriptomic regulation.
慢性缺氧心脏的整合多组学分析:重点关注 m(6)A 和 m(6)Am 表观转录组调控。
Tamoxifen differentially modulates endometrial hyperplasia via wild-type and mutant p53 regulation of the ALKBH5-REG1A axis.
他莫昔芬通过野生型和突变型 p53 对 ALKBH5-REG1A 轴的调控,差异性地调节子宫内膜增生。
Diabetic Kidney Disease Progression Alleviated in Mice by ALKBH5-Mediated UC-MSCs-Derived Exosomes That Inhibit TRAF6 m6A Modification and Promote M2 Macrophage Polarisation.
ALKBH5介导的UC-MSC衍生外泌体抑制TRAF6 m6A修饰并促进M2巨噬细胞极化,从而缓解小鼠糖尿病肾病进展。
ALKBH5-mediated m6A demethylation of TXNDC5 drives malignant progression in gastric cancer.
ALKBH5 介导的 TXNDC5 m6A 去甲基化驱动胃癌的恶性进展。
Identification and validation of PANoptosis-related genes in ankylosing spondylitis.
强直性脊柱炎中PANoptosis相关基因的鉴定和验证。
METTL3/ALKBH5-Mediated N6-Methyladenosine Modification Drives Macrophage M1 Polarization via the SLC15A3-TASL-IRF5 Signaling Axis in Psoriasis.
METTL3/ALKBH5介导的N6-甲基腺苷修饰通过SLC15A3-TASL-IRF5信号轴驱动银屑病巨噬细胞M1极化。
FTO regulates ELK3-mediated metabolic rewiring and represents a unique therapeutic target in T cell leukemia
FTO调控ELK3介导的代谢重编程,是T细胞白血病中一个独特的治疗靶点。
Follicle-stimulating hormone promotes EndMT in endothelial cells by upregulating ALKBH5 expression.
促卵泡激素通过上调ALKBH5表达促进内皮细胞的EndMT
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