Neutralizing antibodies (NAbs) against adeno-associated virus (AAV) represent a significant obstacle to the efficacy of systemic recombinant AAV vector administration or re-administration. While there are some promising preclinical immunomodulation strategies in development, insights into which B cell subsets and compartments maintain persistent AAV NAb may define the optimal eradication strategy. Given the limited success of CD20-directed monotherapy in previous studies, we hypothesized that CD19-directed approaches that extend targeting into the plasma cell compartments may improve AAV NAb eradication. We tested this approach in mice using chimeric antigen receptor T (CAR-T) cells or monoclonal antibodies (mAbs). We observed that combination mAbs targeting CD19, CD22, CD20, or B220 in mice did not eliminate tissue-resident B cells and, correspondingly, did not deplete pre-existing high titer AAV8 NAb. In contrast, CD19 CAR-T therapy eliminated peripheral and tissue-resident B cells and plasma cells and resulted in a marked reduction or eradication of high titer AAV8 NAb that permitted successful transgene expression following systemic AAV8 re-administration in mice. This successful therapeutic approach in mice identifies the population and location of B cells necessary to reduce or eradicate AAV NAb sufficiently to permit successful transgene expression with systemic AAV vector administration.
Use of CD19-targeted immune modulation to eradicate AAV-neutralizing antibodies.
利用 CD19 靶向免疫调节来根除 AAV 中和抗体
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作者:Doshi Bhavya S, Markmann Caroline A, Novak Noelle, Juarez Rojas Silvia, Davidson Robert, Chau Julia Q, Wang Wei, Carrig Sean, Martos Rus Cristina, Samelson-Jones Benjamin J, Small Juliana C, Bhoj Vijay G, George Lindsey A
| 期刊: | Molecular Therapy | 影响因子: | 12.000 |
| 时间: | 2025 | 起止号: | 2025 Jul 2; 33(7):3073-3085 |
| doi: | 10.1016/j.ymthe.2025.03.003 | 研究方向: | 其它 |
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