Ovarian hyperstimulation syndrome (OHSS) is a severe complication of controlled ovarian hyperstimulation (COH) during in vitro fertilization (IVF) treatment, characterized by increased capillary permeability. Vascular endothelial growth factor (VEGF) is a key mediator in OHSS, with serum VEGF levels correlating with its severity. In this study, we investigated the therapeutic potential of (-)-epigallocatechin-3-gallate (EGCG) and its derivative, Pro-EGCG, in mitigating OHSS. Using both in vitro and in vivo models, including primary human granulosa-lutein cells, the human granulosa-like tumor KGN cell line, and a rat OHSS model induced with pregnant mare serum gonadotropin, we found that EGCG and Pro-EGCG significantly reduced OHSS progression. This was supported by histological analyses, reductions in ovarian weight, and decreased VEGF expression at both transcriptomic and proteomic levels. Mechanistic studies revealed that EGCG and Pro-EGCG inhibit TGF-β-induced VEGF production through suppression of the TGF-β/Smad and PKA-CREB signaling pathways. RNA sequencing further validated the downregulation of VEGF expression following treatment. These findings highlight the potential of EGCG as a novel adjuvant therapy for managing OHSS, providing a mechanistic basis for its clinical application.
The Therapeutic Potential of EGCG and Pro-EGCG in Mitigating Ovarian Hyperstimulation Syndrome: Unraveling the Modulatory Mechanism through the VEGF Pathway.
EGCG 和 Pro-EGCG 在缓解卵巢过度刺激综合征中的治疗潜力:通过 VEGF 通路揭示其调节机制
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作者:Wang Sijia, Fang Lanlan, Cong Luping, Jia Qiongqiong, Wu Waner, Zhao Mingpeng, Li Tinchiu, Chung Jacqueline Pui Wah, Fung Ka Kei, Lam Vivian Ching Man, Sun Yingpu, Cheng Jung-Chien, Chan David Yiu Leung
| 期刊: | International Journal of Biological Sciences | 影响因子: | 10.000 |
| 时间: | 2025 | 起止号: | 2025 Apr 22; 21(7):3045-3060 |
| doi: | 10.7150/ijbs.98653 | 研究方向: | 信号转导 |
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