The Combined Role of Silanols and Oxidative Stress in Determining Engineered Stone Dust Toxicity

硅烷醇和氧化应激在决定人造石粉毒性中的联合作用

阅读:2

Abstract

Engineered stone (ES) silicosis is emerging as a global occupational health crisis, caused by exposure to respirable particles generated during the processing of ES composite materials. ES composites comprise crystalline silica (predominantly quartz), inorganic aggregates, polymeric resins, and pigments. The severity of lung disease in workers contrasts with the modest effects observed in short-term in vitro studies, exposing a critical gap in our mechanistic understanding of ES dust toxicity. In this work, we examined the surface chemistry and reactivity of ES dust obtained from a slab with high crystalline silica content, before and after incubation (up to two months) in simulated lung fluids: artificial lysosomal fluid (ALF, pH ∼ 4.5) and lung lining fluid simulant (Gamble's solution, GS, pH ∼ 7.4). Damage to model membranes (red blood cell, RBC), an initiating event in ES-induced toxicity, was quantified by membranolytic assay. Pristine ES dust was negligibly membranolytic. Incubation in ALF markedly increased ES membranolytic activity, correlating with partial degradation of the resin. A complete removal of the resin produced a dust with further enhanced activity, associated with the exposure of nearly free silanol (NFS) groups, a recognized molecular trigger of quartz toxicity. NFS were detected by infrared spectroscopy after H/D isotopic exchange. ALF incubation also led to substantial release of transition metal ions, which catalyzed the formation of hydroxyl and carboxyl radicals, detected by EPR spectroscopy. In contrast, GS exposure resulted in minimal membranolytic activity and low radical generation. Our findings suggest that prolonged residence of ES dust in lung cellular environments, particularly lysosomes, promotes resin degradation, exposes reactive silanols, and releases transition metal ions, thereby imparting both membranolytic and oxidative potential. This work provides new molecular insight into ES dust toxicity, emphasizes the urgency of safer occupational practices, and paves the way to safe-by-design strategies for future composite materials.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。