Leinardi相关文献与解析，生知库 | 链接生命科学的每一步

Pavan, Cristina; Fimiani, Marianna; Cananà, Stefania; Diana, Aleandro; Marafante, Matteo; Bertinetti, Stefano; Escolano-Casado, Guillermo; Mino, Lorenzo; Pisaniello, Dino; Leinardi, Riccardo; Tomatis, Maura; Turci, Francesco

毒理研究

氧化应激

发表日期：2026

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Alveolar macrophages initiate the spatially targeted recruitment of neutrophils after nanoparticle inhalation

吸入纳米颗粒后，肺泡巨噬细胞启动中性粒细胞的空间靶向募集。

期刊:Science Advances

影响因子:11.7

doi:10.1126/sciadv.adx8586

Qiongliang Liu ，Lin Yang ，Chenxi Li ，Lianyong Han ，Juliet Wilmot ，Guo Yang ，Riccardo Leinardi ，Qiaoxia Zhou ，Andreas Schröppel ，David Kutschke ，Judith Secklehner ，Ali Önder Yldirim ，François Huaux ，Dagmar Zeuschner ，Leo M Carlin ，Markus Sperandio ，Otmar Schmid ，Tobias Stoeger ，Markus Rehberg

Uncovering exposome-related diseases through the pathologic metallome: a novel approach for clinical populations

通过病理金属组学揭示暴露组相关疾病：一种适用于临床人群的新方法

期刊:Frontiers in Toxicology

影响因子:4.6

doi:10.3389/ftox.2025.1625874

Leinardi, Riccardo; Huaux, François

发表日期：2025

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Short- and long-term pathologic responses to quartz are induced by nearly free silanols formed during crystal fracturing

石英的短期和长期病理反应是由晶体破裂过程中形成的几乎自由的硅醇引起的

期刊:Particle and Fibre Toxicology

影响因子:7.2

doi:10.1186/s12989-024-00611-8

Cristina Pavan #, Riccardo Leinardi #, Anissa Benhida, Saloua Ibouraadaten, Yousof Yakoub, Sybille van den Brule, Dominique Lison, Francesco Turci, François Huaux

发表日期：2024

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Gasdermin D membrane pores orchestrate IL-1α secretion from necrotic macrophages after NFS-rich silica exposure

富含 NFS 的二氧化硅暴露后，Gasdermin D 膜孔调节坏死巨噬细胞分泌 IL-1α

期刊:Archives of Toxicology

影响因子:4.8

doi:10.1007/s00204-023-03463-x

Riccardo Leinardi, Amandine Pochet, Francine Uwambayinema, Yousof Yakoub, Valérie Quesniaux, Bernhard Ryffel, Petr Broz, Cristina Pavan, François Huaux

Nearly free silanols drive the interaction of crystalline silica polymorphs with membranes: Implications for mineral toxicity

几乎自由的硅醇驱动结晶二氧化硅同质异形体与膜的相互作用：对矿物毒性的影响

期刊:Frontiers in Chemistry

影响因子:3.8

doi:10.3389/fchem.2022.1092221

Cristina Pavan, Guillermo Escolano-Casado, Chiara Bellomo, Stefania Cananà, Maura Tomatis, Riccardo Leinardi, Lorenzo Mino, Francesco Turci

信号转导

发表日期：2023

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Distinct Pro-Inflammatory Mechanisms Elicited by Short and Long Amosite Asbestos Fibers in Macrophages

短和长铁石棉纤维在巨噬细胞中引发的不同促炎机制

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms242015145

Riccardo Leinardi, Jasmine Rita Petriglieri, Amandine Pochet, Yousof Yakoub, Marie Lelong, Alain Lescoat, Francesco Turci, Valérie Lecureur, François Huaux

Molecular recognition between membrane epitopes and nearly free surface silanols explains silica membranolytic activity

膜表位和几乎自由表面硅醇之间的分子识别解释了二氧化硅的膜溶解活性

期刊:Colloids and Surfaces B-Biointerfaces

影响因子:5.4

doi:10.1016/j.colsurfb.2022.112625

Cristina Pavan, Matthew J Sydor, Chiara Bellomo, Riccardo Leinardi, Stefania Cananà, Rebekah L Kendall, Erica Rebba, Marta Corno, Piero Ugliengo, Lorenzo Mino, Andrij Holian, Francesco Turci

信号转导

发表日期：2022

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Think Beyond Particle Cytotoxicity: When Self-Cellular Components Released After Immunogenic Cell Death Explain Chronic Disease Development

超越粒子细胞毒性的思考：免疫原性细胞死亡后释放的自身细胞成分如何解释慢性疾病的发生发展

期刊:Frontiers in Toxicology

影响因子:4.6

doi:10.3389/ftox.2022.887228

Leinardi, Riccardo; Longo Sanchez-Calero, Chiara; Huaux, François

Cytotoxicity of fractured quartz on THP-1 human macrophages: role of the membranolytic activity of quartz and phagolysosome destabilization

断裂石英对THP-1人巨噬细胞的细胞毒性：石英的膜溶解活性和吞噬溶酶体不稳定的作用

期刊:Archives of Toxicology

影响因子:6.9

doi:10.1007/s00204-020-02819-x

Leinardi, Riccardo; Pavan, Cristina; Yedavally, Harita; Tomatis, Maura; Salvati, Anna; Turci, Francesco

The Combined Role of Silanols and Oxidative Stress in Determining Engineered Stone Dust Toxicity

硅烷醇和氧化应激在决定人造石粉毒性中的联合作用

Alveolar macrophages initiate the spatially targeted recruitment of neutrophils after nanoparticle inhalation

吸入纳米颗粒后，肺泡巨噬细胞启动中性粒细胞的空间靶向募集。

Uncovering exposome-related diseases through the pathologic metallome: a novel approach for clinical populations

通过病理金属组学揭示暴露组相关疾病：一种适用于临床人群的新方法

Short- and long-term pathologic responses to quartz are induced by nearly free silanols formed during crystal fracturing

石英的短期和长期病理反应是由晶体破裂过程中形成的几乎自由的硅醇引起的

Gasdermin D membrane pores orchestrate IL-1α secretion from necrotic macrophages after NFS-rich silica exposure

富含 NFS 的二氧化硅暴露后，Gasdermin D 膜孔调节坏死巨噬细胞分泌 IL-1α

Nearly free silanols drive the interaction of crystalline silica polymorphs with membranes: Implications for mineral toxicity

几乎自由的硅醇驱动结晶二氧化硅同质异形体与膜的相互作用：对矿物毒性的影响

Distinct Pro-Inflammatory Mechanisms Elicited by Short and Long Amosite Asbestos Fibers in Macrophages

短和长铁石棉纤维在巨噬细胞中引发的不同促炎机制

Molecular recognition between membrane epitopes and nearly free surface silanols explains silica membranolytic activity

膜表位和几乎自由表面硅醇之间的分子识别解释了二氧化硅的膜溶解活性

Think Beyond Particle Cytotoxicity: When Self-Cellular Components Released After Immunogenic Cell Death Explain Chronic Disease Development

超越粒子细胞毒性的思考：免疫原性细胞死亡后释放的自身细胞成分如何解释慢性疾病的发生发展

Cytotoxicity of fractured quartz on THP-1 human macrophages: role of the membranolytic activity of quartz and phagolysosome destabilization

断裂石英对THP-1人巨噬细胞的细胞毒性：石英的膜溶解活性和吞噬溶酶体不稳定的作用