Altered Nasal Microbiota-Metabolome Interactions in Allergic Rhinitis: Implications for Inflammatory Dysregulation

过敏性鼻炎中鼻腔微生物群-代谢组相互作用的改变:对炎症失调的影响

阅读:1

Abstract

OBJECTIVE: The aim of this study was to investigate the correlation between nasal microbiome, metabolites, and their potential contribution to the pathogenesis of allergic rhinitis (AR), a widespread chronic inflammatory disorder that poses a considerable healthcare burden worldwide. Immune dysregulation and environmental factors are key in the development of allergic responses, but the importance of host-microbiota interactions in influencing these responses is gaining recognition. METHODS: 32 AR patients and 20 healthy controls underwent 16S rDNA sequencing and untargeted metabolomics analysis. Microbial diversity, composition, and functional pathways were compared between groups. Metabolomic alterations were evaluated using LC-MS/MS, and correlations between microbiota and metabolites were analyzed. RESULTS: While α-diversity did not differ significantly between groups, β-diversity analysis revealed distinct microbial community shifts in AR patients. Specifically, Actinobacteria and Bacteroidetes abundances were increased, and genera Vibrio and Aeromonas were significantly enriched. Metabolomic profiling identified 528 differential metabolites, including altered levels of LPC, and pathway analysis highlighted disrupted linoleic acid metabolism, arachidonic acid metabolism, and tryptophan metabolism. Correlation analysis revealed significant associations between specific microbial taxa (eg, Aeromonas, Vibrio) and metabolites (eg, LPC, arachidonic acid), suggesting a potential link between microbiota-derived metabolic shifts and inflammatory responses in AR. CONCLUSION: The perturbation of nasal microbiota-metabolite interactions may play a role in the pathogenesis of AR, emphasizing the need for future investigations into potential pathophysiological mechanisms.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。