Successful Tepotinib Treatment in MET Exon 14 Skipping Mutation-Positive Lung Adenocarcinoma With Idiopathic Pulmonary Fibrosis Through Nintedanib Combination

Tepotinib联合Nintedanib治疗MET外显子14跳跃突变阳性伴特发性肺纤维化的肺腺癌取得成功

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Abstract

The mesenchymal-epithelial transition exon 14 (METex14) skipping mutation is a driver of non-small cell lung cancer (NSCLC). MET tyrosine kinase inhibitors (TKIs), such as tepotinib, are effective against METex14 skipping-positive NSCLC but carry a risk of acute exacerbation of interstitial lung disease (AE-ILD) in patients with idiopathic pulmonary fibrosis (IPF). Here, we present a case of a 54-year-old man with IPF and METex14 skipping-positive lung adenocarcinoma. Following first-line platinum-based chemotherapy, disease control was not achieved for either lung cancer or IPF. Therefore, the patient underwent second-line treatment with tepotinib and the antifibrotic agent nintedanib. This combination effectively controlled both lung cancer and IPF, resulting in a partial response without AE-ILD. Our case suggests that nintedanib treatment may offer a safe and effective option for progressive IPF during MET-TKI therapy, presenting a novel therapeutic approach for patients with lung cancer complicated by IPF.

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