Oral Administration of Clostridium butyricum Alleviates High-Fat Diet-Induced Obesity in Mice by Modulating Gut Akkermansia muciniphila Abundance via Direct Growth Promotion

口服丁酸梭菌可通过直接促进肠道黏液阿克曼菌的生长来调节肠道黏液阿克曼菌的丰度,从而缓解高脂饮食诱导的小鼠肥胖。

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Abstract

This study investigated the anti-obesity effects of Clostridium butyricum (CLB) and the underlying mechanisms through gut microbiota modulation. High-fat diet (HFD)-fed C57BL/6 mice were orally administered CLB (1 × 10(10) CFU/kg/day) for 8 weeks. CLB intervention reduced body weight gain by 13.20% and decreased fat mass (21.10 ± 2.24% vs. HFD: 23.39 ± 2.34%, P < 0.05), while partially restoring fecal butyrate levels (11.73 ± 4.99 vs. HFD: 7.27 ± 3.40 μg/g, P < 0.05). 16S rRNA gene sequencing revealed CLB selectively increased the abundance of Akkermansia, which was depleted in HFD mice. In vitro, butyrate, CLB lysate, and culture supernatant significantly enhanced the growth of Akkermansia muciniphila (AKK), indicating microbial cross-feeding. These findings demonstrate CLB alleviates obesity by restoring the abundance of AKK, partly through the production of metabolites that promote the proliferation of AKK, offering novel insights into probiotic-driven microbiota modulation for metabolic health.

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