Regional Variation in Antioxidant and Anti-Inflammatory Activities of the Brown Alga Sargassum thunbergii and Mechanistic Role of Fucosterol in Inflammation Modulation

褐藻 Sargassum thunbergii 抗氧化和抗炎活性的区域差异及其岩藻甾醇在炎症调节中的机制作用

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Abstract

Background/Objectives: Natural products derived from marine algae serve as promising reservoirs of bioactive compounds for preventing and managing inflammation-associated diseases. This study systematically investigated the regional variations in antioxidant and anti-inflammatory activities of the brown alga Sargassum thunbergii collected from seven coastal regions of Korea and elucidated the underlying mechanisms of action. Methods: Among all samples, the extract from Haeundae-gu exhibited the highest total polyphenol (47.3 ± 2.1 mg GAE/g) and flavonoid (19.8 ± 1.7 mg QE/g) contents, showing superior antioxidant activity in 2,2-diphenyl-1-picrylhydrazyl (DPPH) (RC(50) = 44.54 µg/mL), 2,2'-Azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) (RC(50) = 36.42 µg/mL), and ferric reducing antioxidant power (FRAP) (0.56 mM FeSO(4) eq/mg) assays. In lipopolysaccharide (LPS)-stimulated RAW 264.7 macrophages, the Haeundae-gu extract markedly suppressed nitric oxide (NO) production and downregulated inducible nitric oxide synthase (iNOS) expression in a dose-dependent manner without cytotoxic effects. High-performance liquid chromatography (HPLC) identified fucosterol (10.23 ± 0.17 mg/g extract) as a predominant sterol component, and molecular docking analysis revealed specific hydrogen bonding of fucosterol with TYR489 in the active site of iNOS (GlideScore = -4.774 kcal/mol). Results: These findings indicate that both phenolic compounds and sterols such as fucosterol act synergistically to enhance the antioxidant and anti-inflammatory effects of S. thunbergii. Conclusions: In summary, the mechanistic and functional insights obtained in this study highlight S. thunbergii, particularly from Haeundae-gu, as a promising marine-derived bioresource for developing nutraceuticals and therapeutic interventions against oxidative stress and inflammation-related disorders.

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