Theranostics using (89)Zr/(177)Lu-labeled antibody targeting erythropoietin-producing hepatocellular A2 (EphA2)

利用 (89)Zr/(177)Lu 标记抗体靶向促红细胞生成素肝细胞 A2 (EphA2) 的诊疗一体化

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Abstract

PURPOSE: Erythropoietin-producing hepatocellular A2 (EphA2) is abundantly expressed in various types of cancers. This study aimed to assess the therapeutic effect of (177)Lu-labeled EphA2 antibody in combination with PET imaging using (89)Zr-labeled antibody. METHODS: An EphA2 monoclonal antibody (mAb) (clone 230-1) was labeled with (89)Zr and (177)Lu using DFO and NOTA chelators, respectively. HT-1080 (EphA2-expressing human fibrosarcoma) xenograft mice were intravenously administered [(89)Zr]Zr-EphA2 mAb (n = 6) for PET scanning, and [(177)Lu]Lu-EphA2 mAb (n = 12) to evaluate the biodistribution and specificity of uptake in HT-1080 xenografts in a blocking study. The antitumor effect was evaluated by administering 10 (n = 9) or 3MBq (n = 9) of [(177)Lu]Lu-EphA2 mAb, non-radiolabeled EphA2 mAb (n = 6), [(177)Lu]LuCl(3) solution (10MBq, n = 6), or saline solution (control, n = 4) to HT-1080 xenograft mice. Body weight monitoring and preliminary blood tests were performed to evaluate toxicity. Immunohistochemical staining for EphA2 was performed using HT-1080 xenografts. RESULTS: On [(89)Zr]Zr-EphA2 mAb PET, a markedly high accumulation was observed in the tumor xenograft 1 day after administration (SUVmax, 11.8 ± 4.98), which increased on day 5 (SUVmax, 20.1 ± 6.42). [(177)Lu]Lu-EphA2 mAb exhibited excellent uptake and retention in HT-1080 xenografts (49.4 ± 10.8%IA/g at 24 h and 101.8 ± 26.3%IA/g at 72 h after administration). Normal organs did not show high physiological uptake, except for the blood (16.9 ± 0.43%IA/g at 24 h and 16.1 ± 4.9%IA/g at 72 h). The blocking study demonstrated a significant reduction of tumor uptake 24 h after administration (47.2 ± 8.22 vs. 10.7 ± 0.52%IA/g, P = 0.02). Substantial tumor growth inhibition, and complete remission without severe toxicity (6 of 18 mice), was observed in HT-1080 xenograft mice after [(177)Lu]Lu-EphA2 mAb (3 and 10 MBq) administration. The non-radiolabeled EphA2 mAb showed a minor treatment effect compared with [(177)Lu]Lu-EphA2 mAb. Immunohistochemical staining revealed high EphA2 expression in the HT-1080 xenografts. CONCLUSION: [(89)Zr]Zr-/[(177)Lu]Lu-EphA2 mAb demonstrated high retention in tumors, and [(177)Lu]Lu-EphA2 mAb exhibited marked tumor shrinkage. EphA2 is a promising theranostics target for potential application in various types of cancer.

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