Acute Acalculous Cholecystitis Mimicry: Symptomatic Hepatic Infiltration From Chronic Lymphocytic Leukemia Presenting as Cholestatic Liver Injury

急性非结石性胆囊炎的鉴别诊断:慢性淋巴细胞白血病引起的症状性肝脏浸润表现为胆汁淤积性肝损伤

阅读:1

Abstract

Chronic lymphocytic leukemia (CLL) is the most common adult leukemia in the United States. CLL can infiltrate the liver parenchyma or sinusoids, rarely causing symptomatic hepatic outflow obstruction or acute liver failure, making such complications difficult to recognize. We describe a patient with symptomatic, nonobstructive cholestatic liver injury due to CLL infiltration. A 53-year-old man with CLL diagnosed 4 years prior (Rai stage 0 at diagnosis), managed with observation, presented with 10 days of fever, nausea, vomiting, and diarrhea. Examination was significant for fever (39°C) and right upper quadrant tenderness with no hepatomegaly appreciated on palpation. Laboratory testing demonstrated leukocytosis and a cholestatic-predominant liver injury. Imaging demonstrated gallbladder wall thickening, splenomegaly with diffuse lymphadenopathy, a normal-appearing liver, and no biliary obstruction. Empiric antibiotics did not result in clinical improvement. The hospital course was complicated by worsening leukocytosis and elevation in inflammatory markers. Liver biopsy demonstrated portal and sinusoidal infiltration by small mature lymphocytes, consistent with CLL infiltration. The patient was discharged on hospital day 8 after biochemical and clinical improvement with pulse-dose dexamethasone. Allopurinol was initiated for tumor lysis prophylaxis. Combination therapy with obinutuzumab and venetoclax was initiated for Rai stage III CLL. Symptomatic manifestations of CLL-induced hepatic infiltration are extremely rare. In patients with cholestatic-predominant liver injury and negative biliary imaging, particularly with known CLL or unexplained splenomegaly/adenopathy, malignant hepatic infiltration should be considered early, and liver biopsy can be diagnostic and expedite definitive therapy.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。