Mfsd2a-Targeted Therapy for Ischemic Stroke: Mechanisms, Evidence, and Future Prospects

针对MFSD2A的缺血性卒中靶向治疗:机制、证据和未来展望

阅读:1

Abstract

BACKGROUND: Ischemic stroke remains a major cause of global disability and mortality, with blood-brain barrier (BBB) dysfunction being a pivotal event in its pathology. Major facilitator superfamily domain-containing 2a (Mfsd2a), a key lipid transporter at the BBB, has emerged as a promising yet underexplored therapeutic target. OBJECTIVE: This review proposes a unifying framework that positions Mfsd2a as a central indicator of ischemic stroke pathophysiology and a potential target for treatment. Although direct clinical evidence remains in its early stages, this review synthesizes foundational knowledge from diverse fields. METHODS: We revisit the established biological functions of Mfsd2a, including its role in inhibiting caveolae-mediated transcytosis and transporting omega-3 fatty acids, and detail its core mechanisms in maintaining BBB integrity. This review also correlates these functions with their significant downregulation following ischemic stroke. We then critically evaluate the limited but compelling preclinical evidence from models in which Mfsd2a has been directly targeted and explore innovative therapeutic strategies. Finally, we explicitly address the current limitations, including the scarcity of direct intervention studies, and outline a translational roadmap for future research. RESULTS: By integrating this dispersed evidence chain, this review aims to solidify the theoretical foundation for Mfsd2a-targeted therapies and accelerate their clinical development. CONCLUSION: Targeting Mfsd2a shows a promising therapeutic strategy to protect the BBB and improve neurological outcomes after ischemic stroke.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。