Large-genome phage vB_Eco_ZCEC15 targets gastrointestinal MDR E. coli: evidence from in vitro and Caco-2 cell models

大基因组噬菌体 vB_Eco_ZCEC15 靶向胃肠道耐药大肠杆菌:来自体外和 Caco-2 细胞模型的证据

阅读:1

Abstract

BACKGROUND: Bacterial resistance to traditional antibiotics is spreading at an alarming rate, threatening public health and various industrial applications. Phage therapy has emerged as a promising alternative for combating multidrug-resistant (MDR) bacterial infections. However, most studies have focused on in vitro interactions, often overlooking phage dynamics within human cell environments. METHODOLOGY: In this study, we characterized MDR stool-derived Escherichia coli isolates and assessed their antibiotic resistance profiles. We then isolated, characterized and evaluated the efficacy of bacteriophage vB_Eco_ZCEC15 (ΦZCEC15) against selected strains under optimized culture conditions (pH 7.3) and acidic conditions mimicking the human gastrointestinal tract. To assess host safety, we tested the impact of ΦZCEC15 on Caco-2 colon carcinoma cells. Furthermore, we explored the effect of bacterial lysis by ΦZCEC15 on Caco-2 cell viability to optimize its therapeutic applications. RESULTS: Whole-genome sequencing revealed a large ΦZCEC15 genome (170,313 bp) encoding 272 annotated ORFs, eight tRNAs, and multiple accessory genes. The phage exhibited remarkable stability under diverse physical stressors, including pH 3. Notably, ΦZCEC15 achieved optimal antibacterial activity at a low multiplicity of infection (MOI 0.1), efficiently reducing bacterial titers without compromising eukaryotic host cell viability. In contrast, untreated bacterial infections induced cytotoxic effects in Caco-2 cells. CONCLUSION: These findings highlight the potential of ΦZCEC15 as a safe and effective therapeutic agent against MDR E. coli and encourage the importance of addressing resistance mechanisms to optimize clinical outcomes.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。