UV-A Radiation Impairs Sebaceous-Gland-Related Skin Barrier Function by Inducing Inflammation and Altering Intracellular Sebum-Like Lipid Composition

UV-A辐射通过诱导炎症和改变细胞内皮脂样脂质组成来损害皮脂腺相关的皮肤屏障功能。

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Abstract

BACKGROUND: Sebaceous glands (SGs) secrete sebum to form a protective barrier that maintains skin health. However, exposure to ultraviolet A (UV-A) radiation damages the skin barrier, leading to dryness and inflammation. AIMS: In this study, we investigated how UV-A radiation alters SG function, focusing on inflammation and changes in the composition of intracellular sebum-like lipids. We also explored the role of SG-related mechanisms in UVA-radiation-induced skin barrier damage. METHODS: Human primary sebocytes were cultured from SGs isolated from human skin samples and exposed to different doses of UV-A radiation. Inflammatory cytokines released into the culture medium were measured, followed by gene expression analysis using mRNA extracted from cells to examine specific target genes. Intracellular sebum-like lipid composition was analyzed using liquid chromatography-tandem mass spectrometry, and skin barrier function was evaluated using a three-dimensional reconstructed human epidermis (3D skin) model. RESULTS: UV-A irradiation increased inflammatory cytokine levels in the culture medium and altered the expression levels of several genes. Intracellular sebum-like lipid composition was also modified following UV-A irradiation, with notable sex differences. Furthermore, the skin barrier function was impaired in 3D skin models cultured with the supernatant from UV-A-irradiated sebocyte cultures. CONCLUSIONS: This study demonstrated that UV-A radiation stimulated sebocytes to release inflammatory cytokines and altered gene expression. Additionally, UV-A irradiation modified the intracellular sebum-like lipid composition in a sex-dependent manner, contributing to skin barrier damage.

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