Natural variants of AGL80.5 and FPA.3 contribute to bud break timing in poplar by controlling auxin biosynthesis

AGL80.5 和 FPA.3 的天然变体通过控制生长素的生物合成来影响杨树的萌芽时间。

阅读:2

Abstract

The molecular mechanisms governing phenological characteristics and local adaptation in woody perennials remain largely unexplored. Here, we identified that AGAMOUS-LIKE 80 (PtoAGL80.5) and FLOWERING TIME CONTROL PROTEIN FPA (PtoFPA.3) were simultaneously associated with photoperiod factors and bud break (BB) timing in Populus tomentosa. Transgenic experiments in growth chamber and two latitudinal field trials confirmed their positive roles in promoting BB. Both are activated by the canonical phenological regulator SUPPRESSOR OF OVEREXPRESSION OF CO 1, which, in turn, up-regulates the auxin biosynthesis gene TRYPTOPHAN AMINOTRANSFERASE-RELATED (PtoTAR2.1), accelerating BB via auxin pathway. As superior haplotypes, PtoFPA.3(HAP1) binds PtoAGL80.5(HAP1) messenger RNA to elevate protein, up-regulating PtoTAR2.1 and increasing indole-3-acetic acid accumulation. Under future climate scenarios, frequencies of PtoFPA.3(HAP1) and PtoAGL80.5(HAP1) are predicted to decline, narrowing BB variance across populations. This genetic regulatory network governing BB timing via the auxin pathway in perennial plants provides insights for molecular design breeding and ecological conservation amid climate change.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。