Addition of navitoclax to ruxolitinib for patients with myelofibrosis with progression or suboptimal response

对于病情进展或疗效欠佳的骨髓纤维化患者,可将纳维托克(navitoclax)加入鲁索替尼(ruxolitinib)治疗方案中。

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Abstract

Navitoclax (oral B-cell lymphoma-2 family protein inhibitor induces apoptosis of malignant cells in myelofibrosis (MF). We present pooled cohort 1 results from the phase 2 REFINE trial, which evaluated navitoclax plus ruxolitinib (NAV+RUX) for patients with relapsed/refractory MF with suboptimal response to RUX (≥10 mg twice daily stable dose for ≥12 weeks [cohort 1a] or ≥24 weeks [cohort 1b]). Cohort 1a received add-on NAV 50 mg/d, with escalation to ≤300 mg if platelet count was ≥75 × 10(9)/L. Cohort 1b received NAV 100 or 200 mg/d if platelet count was ≤150 or >150 × 10(9)/L, respectively. The primary end point was spleen volume reduction of ≥35% (SVR(35)) at week 24. Secondary end points included ≥50% total symptoms score (TSS(50)) reduction at week 24, bone marrow fibrosis (BMF) grade changes, anemia response, and safety. In total, 125 patients received ≥1 dose of NAV+RUX. With median follow-up of 21 months, SVR(35) rate was 23% at week 24 and 39% at any time on study (median duration: 11 months). TSS(50) rate was 24% at week 24 and 46% at any time on study. BMF improved by ≥1 grade, any time on study, in 39% of patients. Anemia responses were achieved in 23% of patients. Median overall and progression-free survival were 52.3 and 22.1 months, respectively. No new safety signals were observed. The most common adverse event was thrombocytopenia without clinically significant bleeding. NAV+RUX was tolerable and demonstrated early improvement in disease modification parameters in this difficult-to-treat population. This trial was registered at www.ClinicalTrials.gov as #NCT03222609.

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