Differential copper-guided architectures of amyloid β peptidomimetics modulate oxidation states and catalysis

淀粉样β肽模拟物的差异性铜导向结构调节氧化状态和催化作用

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Abstract

Orchestration of differential architectures of designer peptidomimetics that modulate metal oxidation states to perform multiple chemical transformations remains a challenge. Cu-chelation and self-assembly properties of amyloid β (Aβ14-23) peptide were tuned by the incorporation of cyclic dipeptide (CDP) and pyrene (Py) as the assembly directing and reporting units, respectively. We explore the molecular architectonics of Aβ14-23 derived peptidomimetics (Akd(NMC)Py) to form differential architectures that stabilize distinct Cu oxidation states. The fibrillar self-assembly of Akd(NMC)Py is modulated to form nanosheets by the one-off addition of Cu(II). Notably, the serial addition of Cu(II) resulted in the formation of micelle-like core-shell architectures. The micelle-like and nanosheet architectures were found to differentially stabilize Cu(II) and Cu(I) states and catalyze tandem oxidative-hydrolysis and alkyne-azide cycloaddition reactions, respectively.

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