Hesperidin and Hesperetin from Orange Peel Water Extract Protect against NaIO(3)-Induced Oxidative Damage in Retinal Pigment Epithelial Cells by Modulating PI3K/Akt/HIF-1α/BNIP3 Signaling

橙皮水提取物中的橙皮苷和橙皮素通过调节PI3K/Akt/HIF-1α/BNIP3信号通路保护视网膜色素上皮细胞免受NaIO(3)诱导的氧化损伤

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Abstract

Age-related macular degeneration (AMD) is a leading cause of blindness in the elderly, with oxidative stress a major causative factor. Orange peel, rich in polyphenols and flavonoids, possesses potent antioxidant and anti-inflammatory activities. This study evaluated the protective effects of an ultrasound-assisted aqueous extract (OPWE) of orange peel and its major components, hesperidin and hesperetin, against sodium iodate (NaIO(3))-induced retinal damage. Component identification was performed using 3D-HPLC and LC/MS, and the antioxidant capacity was determined using the DPPH and ABTS assays. In vitro, OPWE reduced oxidative stress, mitochondrial dysfunction, and apoptosis in NaIO(3)-treated ARPE-19 cells through the PI3K/Akt and HIF-1α/BNIP3 pathways. Notably, hesperetin exhibited comparable protective effects to OPWE, restoring cell viability and inhibiting ROS production. In vivo, oral administration of OPWE maintained retinal morphology and function in mice induced by NaIO(3). These findings suggest that OPWE, especially hesperetin, is a promising natural candidate for preventing oxidative stress-related retinal degeneration and maintaining retinal health.

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