From Hit to Lead: Systematic Optimization of the Quinazolinedione Scaffold as Potent and Noncytotoxic Antimalarial Agents

从先导化合物到先导化合物:喹唑啉二酮骨架作为高效无细胞毒性抗疟药的系统优化

阅读:1

Abstract

The emergence of Plasmodium falciparum resistance to artemisinin-based therapies necessitates the urgent discovery of new antimalarials with novel scaffolds and mechanisms. The quinazolinedione scaffold, exemplified by the TCAMS hit TCMDC-125133, was identified as a promising, noncytotoxic starting point. Herein, we report a systematic lead optimization campaign commencing from a simplified and synthetically tractable analogue, compound 9 (IC(50) = 586 nM), which was previously reported by our group. A focused library of 57 novel derivatives was designed and synthesized via a concise 5-step route to systematically explore the structure-activity relationships of the terminal phenyl side chain and the quinazolinedione core. Our investigation revealed two critical findings: (i) the 3,4-difluorophenyl side chain was optimal for potency, and (ii) substitution of the core with small, electron-withdrawing halogens was highly beneficial. This strategy led to the discovery of compound 53 which pairs a 6,7-difluoro core with a 3,4-difluorophenyl side chain demonstrating a potent IC(50) of 116 nM against P. falciparum 3D7. Crucially, this potent analogue and every compound in the series exhibited negligible cytotoxicity against human HepG2 cells (IC(50) > 20 μM) indicating a high selectivity. This work validates the 7-halogenated quinazolinedione scaffold as a promising and selective chemotype for further antimalarial drug development.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。