CD8-guided immunochemotherapy improves pathological complete response rates in high-tumor-burden triple-negative breast cancer

CD8 靶向免疫化疗可提高高肿瘤负荷三阴性乳腺癌的病理完全缓解率

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Abstract

Blockade of the programmed cell death 1 ligand 1 (PD-L1) enhances the efficacy of standard chemotherapy in the neoadjuvant treatment of triple-negative early breast cancer (TNBC) but is associated with adverse events. This phase 2 trial evaluated camrelizumab (anti-PD-1) combined with chemotherapy in locally advanced immunomodulatory TNBC (CD8(+) T cell infiltration ≥10%). From October 2022 to September 2023, 90 stage II-III TNBC patients were randomized to receive neoadjuvant chemotherapy ± camrelizumab (200 mg biweekly). The camrelizumab plus chemotherapy group (n = 45) achieved a pathological complete response (pCR) rate of 62.2% (95% confidence interval [CI]: 46.5%-76.2%) versus 42.2% (95% CI: 27.7%-57.8%) in the chemotherapy-alone group (n = 45), with a 20.0% absolute increase (p = 0.059). Grade ≥3 adverse events occurred in 88.9% (40/45) and 82.2% (37/45) of patients, respectively. Multi-omics analyses demonstrated significantly elevated CD8(+) T cell infiltration in camrelizumab-treated pCR patients, alongside strong correlations between PD-L1 expression, stromal tumor-infiltrating lymphocytes, and CD8(+) density. These results indicate that CD8-guided immunochemotherapy with camrelizumab improves pCR rates in high-tumor-burden TNBC with manageable toxicity, supporting CD8(+) T cell levels as a predictive biomarker for immunotherapy response.

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