Maternal Diabetes Types and the Risk of Pregnancy Loss and Specific Malformations in the Infant

孕妇糖尿病类型与妊娠丢失风险及婴儿特定畸形风险的关系

阅读:1

Abstract

BACKGROUND: To compare the risk of pregnancy loss and the prevalence of specific major congenital malformations (MCMs) in newborns between mothers with pregestational diabetes mellitus (DM) type 1 (T1DM) and type 2 (T2DM). METHODS: We used the Merative MarketScan Commercial Claims and Encounters Database (2011-2021) to identify pregnancies among women enrolled from 180 days before to 280 days after the last menstrual period. Diabetes types, pregnancy losses, and MCMs diagnosed within 90 days after birth were identified using validated algorithms. Antidiabetic treatments were used as a proxy for T2DM progression. Analyses of MCMs were restricted to live birth pregnancies linked to infants. RESULTS: Of 2,090,452 eligible pregnancies, 4584 had T1DM and 14,156 had T2DM diagnoses recorded. The spontaneous abortion risk was 17.5% for mothers with T1DM, 21.7% for mothers with T2DM, and 15.9% for mothers without DM. The stillbirth risk was 0.8%, 0.9%, and 0.4%, respectively. The largest adjusted odds ratios relative to no DM were for cardiac malformations for both T1DM (2.79, 95% CI 2.13-3.64) and T2DM (2.02, 95% CI 1.74-2.35). The prevalence of central nervous system, urinary, gastrointestinal, and musculoskeletal malformations was also elevated for both DM types; although they were slightly more frequent in the offspring of mothers with T1DM (which is mostly insulin dependent) compared to T2DM. Among mothers with T2DM, MCM risk was highest in those on insulin therapy. CONCLUSIONS: Relative to mothers without pregestational DM, mothers with T2DM had higher risks of spontaneous abortion, and mothers with T1DM or T2DM had a higher risk of stillbirths and a higher prevalence of many different MCMs in their offspring. Within patients with T2DM, the risk of MCM depended on disease progression.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。