Small Cell Transformation of Metachronous ALK + ve Lung and Prostatic Adenocarcinoma following Initial Response to Ceritinib and Androgen Deprivation Therapies and Metastases to Two Different Organs: Guidance through FDG-PET/CT and (68) Ga-PSMA-11 PET-CT and Biopsy-Immunohistochemistry Correlation

ALK阳性异时性肺癌和前列腺腺癌在接受色瑞替尼和雄激素剥夺疗法初始治疗后发生小细胞转化并转移至两个不同器官:FDG-PET/CT和(68)Ga-PSMA-11 PET-CT引导及活检-免疫组化相关性分析

阅读:1

Abstract

Metachronous malignancies carry poor prognosis and pose certain challenges for management of two different malignancies simultaneously. A 59-year-old male, a patient of ALK+ lung adenocarcinoma demonstrating excellent initial response to targeted therapy with ALK inhibitor ceritinib, developed prostatic adenocarcinoma with neuroendocrine differentiation 36 months later, which was treated with abiraterone and leuprolide. Dual-tracer positron emission tomography (PET) imaging with (18) F-FDG-PET/computed tomography (CT) and (68) Ga-PSMA-11 PET/CT showed complete response of prostatic adenocarcinoma with controlled serum prostate-specific antigen level, showed new-onset metastatic brain and liver lesions 12 months later, the biopsy of the later revealed metastatic small cell neoplasia. The clinical profile, dual tracer PET/CT and immunohistochemistry correlation assisted in concluding that prostate adenocarcinoma had transformed into small cell type, which metastasized to liver, whereas the lung adenocarcinoma had metastasized to brain.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。