日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Histone modification-regulated LncRNA DLEU1 interacts with ASCC2/ALKBH3 complex to drive DNA repair, antioxidant homeostasis and glucose metabolism in gastric cancer.

组蛋白修饰调控的长链非编码RNA DLEU1与ASCC2/ALKBH3复合物相互作用,驱动胃癌中的DNA修复、抗氧化稳态和葡萄糖代谢。

Zhang Xiaoyan, Wang Xin, Wang Qi, Wang Xu, Sun Hui, Liu Yingxue, Tan Cong, Ni Shujuan, Weng Weiwei, Zhang Meng, Wang Lei, Huang Dan, Chen Jie, Wang Xiaoyu, Gan Lu, Abudurexiti Mierxiati, Wang Wenfeng, Chang Jinjia, Sheng Weiqi, Xu Midie

A comprehensive functional atlas of ALK kinase domain variants reveals resistance landscape to ALK inhibitors

ALK激酶结构域变体的综合功能图谱揭示了ALK抑制剂的耐药性图谱

Oh, Hyeong-Cheol; Han, Yeonseung; Chang, Yoojin; Kim, Hyongbum Henry

Insights into the m(6)A demethylases FTO and ALKBH5 : structural, biological function, and inhibitor development

对m(6)A去甲基化酶FTO和ALKBH5的深入研究:结构、生物学功能和抑制剂开发

Gao, Zewei; Zha, Xuan; Li, Min; Xia, Xueli; Wang, Shengjun

Captive ERVWE1 triggers impairment of 5-HT neuronal plasticity in the first-episode schizophrenia by post-transcriptional activation of HTR1B in ALKBH5-m6A dependent epigenetic mechanisms

捕获的ERVWE1通过ALKBH5-m6A依赖的表观遗传机制,在转录后激活HTR1B,从而引发首发精神分裂症中5-HT神经元可塑性的损伤。

Wu, Xiulin; Liu, Lianzhong; Xue, Xing; Li, Xuhang; Zhao, Kexin; Zhang, Jiahang; Li, Wenshi; Yao, Wei; Ding, Shuang; Jia, Chen; Zhu, Fan

EML4-ALK Variant-Specific Genetic Interactions Shape Lung Tumorigenesis

EML4-ALK 变异特异性基因相互作用影响肺肿瘤发生

Alberto Diaz-Jimenez #,Emily G Shuldiner #,Kalman Somogyi,Karen Y Shih,Óscar González-Velasco,Mulham Najajreh,Stewart Kim,Filiz Akkas,Christopher W Murray,Laura Andrejka,Min K Tsai,Benedikt Brors,Ilse Hofmann,Smruthy Sivakumar,Saumya D Sisoudiya,Ethan S Sokol,Hongchen Cai,Dmitri A Petrov,Monte M Winslow,Rocio Sotillo

Jaye DL, Feldman AL. ALK-negative anaplastic large cell lymphoma with JAK2 rearrangement mimicking classic Hodgkin lymphoma. Blood. 2023;141(17):2160

Jaye DL, Feldman AL. ALK阴性间变性大细胞淋巴瘤伴JAK2重排,类似经典霍奇金淋巴瘤。Blood. 2023;141(17):2160

ALKB-1-dependent tRNA methylation is required for efficient paternal mitochondrial elimination.

ALKB-1依赖的tRNA甲基化是有效清除父系线粒体所必需的。

Reversible ALKBH5 cytosolic aggregation accelerates cellular senescence.

ALKBH5胞质可逆聚集加速细胞衰老。

Tendon Organoids Enable Functional Tendon Rejuvenation Through ALKBH5-Dependent RNA Demethylation.

肌腱类器官通过 ALKBH5 依赖性 RNA 去甲基化实现肌腱功能性再生。

Qin Tian, Pan Aini, Miao Zhuoning, Zhao Yanyan, Mo Xianan, Sun Heng, Fan Chunmei, Guo Junyu, Wu Bingbing, Shen Weiliang, Zheng Qiangqiang, Lu Jun, Jiang Xi, Yin Zi, Chen Xiao

Site-Specific Mitochondrial RNA N1-Methyladenosine Demethylation via an Engineered MTS-PUF-ALKBH3 Fusion Protein.

通过工程化的 MTS-PUF-ALKBH3 融合蛋白进行位点特异性线粒体 RNA N1-甲基腺苷去甲基化。