Baro Marta相关文献与解析，生知库 | 链接生命科学的每一步

Kelley, Vanessa M; Baro, Marta; Gasperi, William E; Ader, Nicholas R; Lea, Hannah; Lee, Hojin; Phoomak, Chatchai; Kabeche, Lilian; King, Megan C; Contessa, Joseph N

JAK/STAT

细胞生物学

JAK1

发表日期：2026

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Redundancy of the OST catalytic subunit facilitates therapeutic targeting of N-glycosylation

OST催化亚基的冗余性有利于以N-糖基化为治疗靶点。

期刊:Cell Chemical Biology

影响因子:7.2

doi:10.1016/j.chembiol.2025.05.005

Baro, Marta; Lee, Hojin; Kelley, Vanessa; Lou, Rongliang; Phoomak, Chatchai; Politi, Katerina; Zeiss, Caroline J; Van Zandt, Michael; Contessa, Joseph N

发表日期：2025

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PLK4 Inhibition as a Strategy to Enhance Non-Small Cell Lung Cancer Radiosensitivity.

PLK4抑制作为增强非小细胞肺癌放射敏感性的策略

期刊:Molecular Cancer Therapeutics

影响因子:5.5

doi:10.1158/1535-7163.MCT-24-0978

DomÃnguez-Vigil Irma G, Banik Kishore, Baro Marta, Contessa Joseph N, Hayman Thomas J

Loss of JAK1 Function Causes G2/M Cell Cycle Defects Vulnerable to Kif18a Inhibition.

JAK1 功能丧失导致 G2/M 细胞周期缺陷，易受 Kif18a 抑制

期刊:

影响因子:

doi:10.1101/2025.02.19.638911

Kelley Vanessa, Baro Marta, Gasperi William, Ader Nicholas, Lea Hannah, Lee Hojin, Phoomak Chatchai, Kabeche Lilian, King Megan, Contessa Joseph

细胞生物学

发表日期：2025

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Cetuximab may inhibit tumor growth and angiogenesis induced by ionizing radiation: a preclinical rationale for maintenance treatment after radiotherapy

西妥昔单抗可能抑制电离辐射诱导的肿瘤生长和血管生成：放疗后维持治疗的临床前理论依据

期刊:Oncologist

影响因子:4.2

doi:10.1634/theoncologist.2008-0290

Pueyo, Gemma; Mesia, Ricard; Figueras, Agnes; Lozano, Alicia; Baro, Marta; Vazquez, Silvia; Capella, Gabriel; Balart, Josep

Loss of JAK1 Function Causes G2/M Cell Cycle Defects Vulnerable to KIF18A Inhibition

JAK1功能丧失导致G2/M期细胞周期缺陷，该缺陷易受KIF18A抑制的影响

Redundancy of the OST catalytic subunit facilitates therapeutic targeting of N-glycosylation

OST催化亚基的冗余性有利于以N-糖基化为治疗靶点。

PLK4 Inhibition as a Strategy to Enhance Non-Small Cell Lung Cancer Radiosensitivity.

PLK4抑制作为增强非小细胞肺癌放射敏感性的策略

Loss of JAK1 Function Causes G2/M Cell Cycle Defects Vulnerable to Kif18a Inhibition.

JAK1 功能丧失导致 G2/M 细胞周期缺陷，易受 Kif18a 抑制

Cetuximab may inhibit tumor growth and angiogenesis induced by ionizing radiation: a preclinical rationale for maintenance treatment after radiotherapy

西妥昔单抗可能抑制电离辐射诱导的肿瘤生长和血管生成：放疗后维持治疗的临床前理论依据