日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Patient-Derived 3D Bioprinted Cardiac Organoid Constructs Reveal Key Pathological Features of Duchenne Muscular Dystrophy

患者来源的3D生物打印心脏类器官构建体揭示杜氏肌营养不良症的关键病理特征

Marini, Vittoria; Campaner Socias, Margalida; Dimopoulos, Andreas; Rinvenuto, Lorenza; Pozzo, Enrico; Stalkopf, Diana; Serafini, Angelo; Morri, Sara; Wang, Ashley; La Rovere, Rita; Chai, Yoke Chin; Bollini, Sveva; Bultynck, Geert; Roderick, H Llewelyn; Papantoniou, Ioannis; Sampaolesi, Maurilio

The BCL2 family: from apoptosis mechanisms to new advances in targeted therapy

BCL2家族:从细胞凋亡机制到靶向治疗的新进展

Vogler, Meike; Braun, Yannick; Smith, Victoria M; Westhoff, Mike-Andrew; Pereira, Raquel S; Pieper, Nadja M; Anders, Marius; Callens, Manon; Vervliet, Tim; Abbas, Maha; Macip, Salvador; Schmid, Ralf; Bultynck, Geert; Dyer, Martin Js

Author Correction: Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis

作者更正:内质网-线粒体接触是磷脂过氧化的主要热点区域,可驱动铁死亡。

Sassano, Maria Livia; Tyurina, Yulia Y; Diokmetzidou, Antigoni; Vervoort, Ellen; Tyurin, Vladimir A; More, Sanket; La Rovere, Rita; Giordano, Francesca; Bultynck, Geert; Pavie, Benjamin; Swinnen, Johan V; Bayir, Hülya; Kagan, Valerian E; Scorrano, Luca; Agostinis, Patrizia

Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis.

内质网-线粒体接触是磷脂过氧化的主要热点,可导致铁死亡

Sassano Maria Livia, Tyurina Yulia Y, Diokmetzidou Antigoni, Vervoort Ellen, Tyurin Vladimir A, More Sanket, La Rovere Rita, Giordano Francesca, Bultynck Geert, Pavie Benjamin, Swinnen Johan V, Bayir Hülya, Kagan Valerian E, Scorrano Luca, Agostinis Patrizia

Correction to: Disruption of MAM integrity in mutant FUS oligodendroglial progenitors from hiPSCs

更正:hiPSCs来源的突变型FUS少突胶质细胞祖细胞中MAM完整性的破坏

Zhu, Yingli; Burg, Thibaut; Neyrinck, Katrien; Vervliet, Tim; Nami, Fatemeharefeh; Vervoort, Ellen; Ahuja, Karan; Sassano, Maria Livia; Chai, Yoke Chin; Tharkeshwar, Arun Kumar; De Smedt, Jonathan; Hu, Haibo; Bultynck, Geert; Agostinis, Patrizia; Swinnen, Johannes V; Van Den Bosch, Ludo; da Costa, Rodrigo Furtado Madeiro; Verfaillie, Catherine

10th European Calcium Society symposium: The Ca2+-signaling toolkit in cell function, health and disease

第十届欧洲钙学会研讨会:Ca2+信号通路在细胞功能、健康和疾病中的作用

Speelman-Rooms, Femke; Vanmunster, Maarten; Coughlan, Aled; Hinrichs, Macarena; Pontisso, Ilaria; Barbeau, Solene; Parpaite, Thibaud; Bultynck, Geert; Brohus, Malene

Author Correction: Disrupted Ca(2+) homeostasis and immunodeficiency in patients with functional IP(3) receptor subtype 3 defects

作者更正:功能性 IP(3) 受体亚型 3 缺陷患者的 Ca(2+) 稳态紊乱和免疫缺陷

Neumann, Julika; Van Nieuwenhove, Erika; Terry, Lara E; Staels, Frederik; Knebel, Taylor R; Welkenhuyzen, Kirsten; Ahmadzadeh, Kourosh; Baker, Mariah R; Gerbaux, Margaux; Willemsen, Mathijs; Barber, John S; Serysheva, Irina I; De Waele, Liesbeth; Vermeulen, François; Schlenner, Susan; Meyts, Isabelle; Yule, David I; Bultynck, Geert; Schrijvers, Rik; Humblet-Baron, Stephanie; Liston, Adrian

Connexin hemichannels as candidate targets for cardioprotective and anti-arrhythmic treatments

连接蛋白半通道作为心脏保护和抗心律失常治疗的候选靶点

Leybaert, Luc; De Smet, Maarten Aj; Lissoni, Alessio; Allewaert, Rosalie; Roderick, H Llewelyn; Bultynck, Geert; Delmar, Mario; Sipido, Karin R; Witschas, Katja

Loss of INPP5K attenuates IP(3)-induced Ca(2+) responses in the glioblastoma cell line U-251 MG cells.

INPP5K 的缺失会减弱胶质母细胞瘤细胞系 U-251 MG 细胞中 IP(3) 诱导的 Ca(2+) 反应

Loncke Jens, Luyten Tomas, Ramos Ana Raquel, Erneux Christophe, Bultynck Geert

Dysregulated Ca(2+) Homeostasis as a Central Theme in Neurodegeneration: Lessons from Alzheimer's Disease and Wolfram Syndrome

钙离子稳态失调是神经退行性疾病的核心主题:来自阿尔茨海默病和沃尔夫拉姆综合征的启示

Callens, Manon; Loncke, Jens; Bultynck, Geert