Burke Thomas P相关文献与解析，生知库 | 链接生命科学的每一步

Differences between human and rodent nitric oxide production dictate susceptibility to tick-borne Rickettsia

人类和啮齿动物一氧化氮生成量的差异决定了它们对蜱传立克次体的易感性。

期刊:

影响因子:

doi:10.1101/2025.06.27.661835

Luu, Anh Phuong; Guzman, Alejandro Amando; Bouin, Alexis; Drayman, Nir; Burke, Thomas P

发表日期：2025

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RECON-Dependent Inflammation in Hepatocytes Enhances Listeria monocytogenes Cell-to-Cell Spread

肝细胞中RECON依赖性炎症增强单核细胞增生李斯特菌的细胞间传播。

期刊:mBio

影响因子:4.7

doi:10.1128/mBio.00526-18

McFarland, Adelle P; Burke, Thomas P; Carletti, Alexie A; Glover, Rochelle C; Tabakh, Hannah; Welch, Matthew D; Woodward, Joshua J

Immunology/Inflammation

发表日期：2018

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Listeria monocytogenes is resistant to lysozyme through the regulation, not the acquisition, of cell wall-modifying enzymes

单核细胞增生李斯特菌对溶菌酶的抵抗力是通过调节而非获得细胞壁修饰酶来实现的。

期刊:Journal of Bacteriology

影响因子:3

doi:10.1128/JB.02053-14

Burke, Thomas P; Loukitcheva, Anastasia; Zemansky, Jason; Wheeler, Richard; Boneca, Ivo G; Portnoy, Daniel A

Listeria monocytogenes engineered to activate the Nlrc4 inflammasome are severely attenuated and are poor inducers of protective immunity

经基因工程改造以激活Nlrc4炎症小体的单核细胞增生李斯特菌毒力显著减弱，且诱导保护性免疫的能力较弱。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.1019041108

Sauer, John-Demian; Pereyre, Sabine; Archer, Kristina A; Burke, Thomas P; Hanson, Bill; Lauer, Peter; Portnoy, Daniel A

Host glutathione is required for Rickettsia parkeri cell division and intracellular survival.

宿主谷胱甘肽是帕克氏立克次体细胞分裂和细胞内生存所必需的

Correction: Interferon receptor-deficient mice are susceptible to eschar-associated rickettsiosis

更正：干扰素受体缺陷小鼠易感焦痂相关立克次体病

Differences between human and rodent nitric oxide production dictate susceptibility to tick-borne Rickettsia

人类和啮齿动物一氧化氮生成量的差异决定了它们对蜱传立克次体的易感性。

RECON-Dependent Inflammation in Hepatocytes Enhances Listeria monocytogenes Cell-to-Cell Spread

肝细胞中RECON依赖性炎症增强单核细胞增生李斯特菌的细胞间传播。

Listeria monocytogenes is resistant to lysozyme through the regulation, not the acquisition, of cell wall-modifying enzymes

单核细胞增生李斯特菌对溶菌酶的抵抗力是通过调节而非获得细胞壁修饰酶来实现的。

Listeria monocytogenes engineered to activate the Nlrc4 inflammasome are severely attenuated and are poor inducers of protective immunity

经基因工程改造以激活Nlrc4炎症小体的单核细胞增生李斯特菌毒力显著减弱，且诱导保护性免疫的能力较弱。