日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

A Snapshot of the Global Race for Vaccines Targeting SARS-CoV-2 and the COVID-19 Pandemic

全球 SARS-CoV-2 和 COVID-19 疫苗研发竞赛一瞥

Colin D Funk, Craig Laferrière, Ali Ardakani

Perivascular adipose tissue-derived extracellular vesicle miR-221-3p mediates vascular remodeling

血管周围脂肪组织来源的细胞外囊泡 miR-221-3p 介导血管重塑

Xinzhi Li ,Laurel L Ballantyne ,Ying Yu ,Colin D Funk

Transplantation of Gene-Edited Hepatocyte-like Cells Modestly Improves Survival of Arginase-1-Deficient Mice

基因编辑的肝细胞样细胞移植可轻微提高精氨酸酶-1 缺陷小鼠的生存率

Yuan Yan Sin, Laurel L Ballantyne, Christopher R Richmond, Colin D Funk

Flipping the cyclooxygenase (Ptgs) genes reveals isoform-specific compensatory functions

翻转环氧合酶 (Ptgs) 基因揭示了异构体特异性的补偿功能

Xinzhi Li, Liudmila L Mazaleuskaya, Chong Yuan, Laurel L Ballantyne, Hu Meng, William L Smith, Garret A FitzGerald, Colin D Funk

Genomic and lipidomic analyses differentiate the compensatory roles of two COX isoforms during systemic inflammation in mice

基因组学和脂质组学分析区分了小鼠全身炎症过程中两种 COX 亚型的补偿作用

Xinzhi Li, Liudmila L Mazaleuskaya, Laurel L Ballantyne, Hu Meng, Garret A FitzGerald, Colin D Funk

Isoform-Specific Compensation of Cyclooxygenase (Ptgs) Genes during Implantation and Late-Stage Pregnancy

环氧合酶 (Ptgs) 基因在植入和妊娠晚期的异构体特异性补偿

Xinzhi Li, Laurel L Ballantyne, Mackenzie C Crawford, Garret A FitzGerald, Colin D Funk

Proof-of-Concept Gene Editing for the Murine Model of Inducible Arginase-1 Deficiency

针对可诱导精氨酸酶-1 缺乏症小鼠模型的概念验证基因编辑

Yuan Yan Sin, Phillipe R Price, Laurel L Ballantyne, Colin D Funk

Effects of p53-knockout in vascular smooth muscle cells on atherosclerosis in mice

血管平滑肌细胞p53基因敲除对小鼠动脉粥样硬化的影响

Richard Yang Cao, Robert Eves, Lilly Jia, Colin D Funk, Zongchao Jia, Alan S Mak

Liver-specific knockout of arginase-1 leads to a profound phenotype similar to inducible whole body arginase-1 deficiency

肝脏特异性敲除精氨酸酶-1 可导致与可诱导全身精氨酸酶-1 缺乏症相似的表型

Laurel L Ballantyne, Yuan Yan Sin, Osama Y Al-Dirbashi, Xinzhi Li, David J Hurlbut, Colin D Funk

Endogenously generated omega-3 fatty acids attenuate vascular inflammation and neointimal hyperplasia by interaction with free fatty acid receptor 4 in mice

内源性生成的ω-3脂肪酸通过与小鼠体内的游离脂肪酸受体4相互作用,减轻血管炎症和新生内膜增生。

Xinzhi Li ,Laurel L Ballantyne ,Xinghui Che ,Jeffrey D Mewburn ,Jing X Kang ,Robert M Barkley ,Robert C Murphy ,Ying Yu ,Colin D Funk