5'-Methylthioadenosine Metabolic Reprogramming Drives H3K79 Monomethylation-Mediated PAK2 Upregulation to Promote Cadmium-Induced Breast Cancer Progression by Impairing Autophagic Flux.
5'-甲基硫代腺苷代谢重编程驱动H3K79单甲基化介导的PAK2上调，通过损害自噬流促进镉诱导的乳腺癌进展。
期刊:Advanced Science
影响因子:14.1
doi:10.1002/advs.202500941
Li Jingdian, Deng Ping, Fan Tengfei, Qu Yuchen, Tan Miduo, Liang Yidan, Gao Peng, Peng Yongchun, Qin Mingke, Jie Sheng, Hao Rongrong, Wang Liting, Zhang Lei, Chen Chunhai, He Mindi, Ma Qinlong, Luo Yan, Tian Li, Xie Jia, Chen Mengyan, Tian Rui, Li Min, Yu Zhengping, Zhou Zhou, Pi Huifeng
肿瘤
表观遗传
肿瘤免疫
乳腺癌
代谢
自噬流
代谢重编程
PAK2
自噬
