Denise M Inman相关文献与解析，生知库 | 链接生命科学的每一步

Long-term HIF-1α stabilization reduces respiration, promotes mitophagy, and results in retinal cell death

长期 HIF-1α 稳定会降低呼吸作用、促进线粒体自噬并导致视网膜细胞死亡

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-023-47942-8

Nana Yaa Nsiah, Autumn B Morgan, Nina Donkor, Denise M Inman

Destabilizing COXIV in Müller Glia Increases Retinal Glycolysis and Alters Scotopic Electroretinogram

穆勒胶质细胞中 COXIV 不稳定会增加视网膜糖酵解并改变暗视视网膜电图

期刊:Cells

影响因子:5.1

doi:10.3390/cells11233756

Nana Yaa Nsiah, Denise M Inman

Stretch stress propels glutamine dependency and glycolysis in optic nerve head astrocytes

拉伸应力促进视神经乳头星形胶质细胞中的谷氨酰胺依赖性和糖酵解

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2022.957034

Nathaniel Pappenhagen, Eric Yin, Autumn B Morgan, Charles C Kiehlbauch, Denise M Inman

Ocular Hypertension Results in Hypoxia within Glia and Neurons throughout the Visual Projection

眼压升高导致整个视觉投射过程中的神经胶质细胞和神经元缺氧

期刊:Antioxidants

影响因子:

doi:10.3390/antiox11050888

Assraa Hassan Jassim, Nana Yaa Nsiah, Denise M Inman

Oxidative Stress and Hypoxia Modify Mitochondrial Homeostasis During Glaucoma

氧化应激和缺氧改变青光眼期间的线粒体稳态

期刊:Antioxidants & Redox Signaling

影响因子:5.9

doi:10.1089/ars.2020.8180

Assraa Hassan Jassim, Yan Fan, Nathaniel Pappenhagen, Nana Yaa Nsiah, Denise M Inman

Transcorneal Electrical Stimulation Reduces Neurodegenerative Process in a Mouse Model of Glaucoma

经角膜电刺激可减缓青光眼小鼠模型中的神经退行性过程

期刊:Annals of Biomedical Engineering

影响因子:3

doi:10.1007/s10439-020-02608-8

Assraa Hassan Jassim #, McKay Cavanaugh #, Jessica Stukel Shah, Rebecca Willits, Denise M Inman

MCT2 overexpression rescues metabolic vulnerability and protects retinal ganglion cells in two models of glaucoma

MCT2 过表达可挽救两种青光眼模型中的代谢脆弱性并保护视网膜神经节细胞

期刊:Neurobiology of Disease

影响因子:5.1

doi:10.1016/j.nbd.2020.104944

Mohammad Harun-Or-Rashid, Nathaniel Pappenhagen, Ryan Zubricky, Lucy Coughlin, Assraa Hassan Jassim, Denise M Inman

Evidence of Hypoxic Glial Cells in a Model of Ocular Hypertension

眼高压模型中缺氧胶质细胞的证据

期刊:Investigative Ophthalmology & Visual Science

影响因子:5

doi:10.1167/iovs.18-24977

Assraa H Jassim, Denise M Inman

Higher Reliance on Glycolysis Limits Glycolytic Responsiveness in Degenerating Glaucomatous Optic Nerve

对糖酵解的依赖性较高会限制退化性青光眼视神经的糖酵解反应性

期刊:Molecular Neurobiology

影响因子:4.6

doi:10.1007/s12035-019-1576-4

Assraa Hassan Jassim, Lucy Coughlin, Mohammad Harun-Or-Rashid, Patrick T Kang, Yeong-Renn Chen, Denise M Inman

神经

发表日期：2019

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The ketogenic diet and hypoxia promote mitophagy in the context of glaucoma

生酮饮食和缺氧在青光眼的背景下促进线粒体自噬

Long-term HIF-1α stabilization reduces respiration, promotes mitophagy, and results in retinal cell death

长期 HIF-1α 稳定会降低呼吸作用、促进线粒体自噬并导致视网膜细胞死亡

Destabilizing COXIV in Müller Glia Increases Retinal Glycolysis and Alters Scotopic Electroretinogram

穆勒胶质细胞中 COXIV 不稳定会增加视网膜糖酵解并改变暗视视网膜电图

Stretch stress propels glutamine dependency and glycolysis in optic nerve head astrocytes

拉伸应力促进视神经乳头星形胶质细胞中的谷氨酰胺依赖性和糖酵解

Ocular Hypertension Results in Hypoxia within Glia and Neurons throughout the Visual Projection

眼压升高导致整个视觉投射过程中的神经胶质细胞和神经元缺氧

Oxidative Stress and Hypoxia Modify Mitochondrial Homeostasis During Glaucoma

氧化应激和缺氧改变青光眼期间的线粒体稳态

Transcorneal Electrical Stimulation Reduces Neurodegenerative Process in a Mouse Model of Glaucoma

经角膜电刺激可减缓青光眼小鼠模型中的神经退行性过程

MCT2 overexpression rescues metabolic vulnerability and protects retinal ganglion cells in two models of glaucoma

MCT2 过表达可挽救两种青光眼模型中的代谢脆弱性并保护视网膜神经节细胞

Evidence of Hypoxic Glial Cells in a Model of Ocular Hypertension

眼高压模型中缺氧胶质细胞的证据

Higher Reliance on Glycolysis Limits Glycolytic Responsiveness in Degenerating Glaucomatous Optic Nerve

对糖酵解的依赖性较高会限制退化性青光眼视神经的糖酵解反应性