Hideki Kitaura相关文献与解析，生知库 | 链接生命科学的每一步

Exogenous Angiotensin-(1-7) Provides Protection Against Inflammatory Bone Resorption and Osteoclastogenesis by Inhibition of TNF-α Expression in Macrophages

外源性血管紧张素-(1-7) 通过抑制巨噬细胞中的 TNF-α 表达来防止炎症性骨吸收和破骨细胞生成

Jiayi Ren, Hideki Kitaura, Takahiro Noguchi, Fumitoshi Ohori, Aseel Marahleh, Jinghan Ma, Kayoko Kanou, Ziqiu Fan, Itaru Mizoguchi

细胞生物学

巨噬细胞

发表日期：2024

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(D-Ala2)GIP Inhibits Inflammatory Bone Resorption by Suppressing TNF-α and RANKL Expression and Directly Impeding Osteoclast Formation

(D-Ala2)GIP 通过抑制 TNF-α 和 RANKL 表达并直接阻碍破骨细胞形成来抑制炎症性骨吸收

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms25052555

Angyi Lin, Hideki Kitaura, Fumitoshi Ohori, Takahiro Noguchi, Aseel Marahleh, Jinghan Ma, Jiayi Ren, Mariko Miura, Ziqiu Fan, Kohei Narita, Itaru Mizoguchi

Fermented Rice Bran Supplementation Inhibits LPS-Induced Osteoclast Formation and Bone Resorption in Mice

发酵米糠补充剂可抑制小鼠 LPS 诱导的破骨细胞形成和骨吸收

期刊:Nutrients

影响因子:4.8

doi:10.3390/nu15133044

Takahiro Noguchi, Hideki Kitaura, Aseel Marahleh, Afifah Zahra Agista, Yusuke Ohsaki, Hitoshi Shirakawa, Itaru Mizoguchi

Generating Bone Marrow Chimeric Mouse Using GPR120 Deficient Mouse for the Study of DHA Inhibitory Effect on Osteoclast Formation and Bone Resorption

利用GPR120缺陷小鼠制备骨髓嵌合小鼠，研究DHA对破骨细胞形成和骨吸收的抑制作用

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms242317000

Jinghan Ma, Hideki Kitaura, Fumitoshi Ohori, Takahiro Noguchi, Aseel Marahleh, Ria Kinjo, Kayoko Kanou, Jiayi Ren, Mariko Miura, Kohei Narita, Itaru Mizoguchi

Azilsartan inhibits inflammation-triggered bone resorption and osteoclastogenesis in vivo via suppression of TNF-α expression in macrophages

阿齐沙坦通过抑制巨噬细胞中的 TNF-α 表达来抑制体内炎症引发的骨吸收和破骨细胞生成

期刊:Frontiers in Endocrinology

影响因子:

doi:10.3389/fendo.2023.1207502

Ziqiu Fan, Hideki Kitaura, Jiayi Ren, Fumitoshi Ohori, Takahiro Noguchi, Aseel Marahleh, Jinghan Ma, Kayoko Kanou, Mariko Miura, Kohei Narita, Angyi Lin, Itaru Mizoguchi

Salt-Sensitive Hypertension Induces Osteoclastogenesis and Bone Resorption via Upregulation of Angiotensin II Type 1 Receptor Expression in Osteoblasts

盐敏感性高血压通过上调成骨细胞中血管紧张素 II 1 型受体的表达来诱导破骨细胞生成和骨吸收

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.6

doi:10.3389/fcell.2022.816764

Adya Pramusita, Hideki Kitaura, Fumitoshi Ohori, Takahiro Noguchi, Aseel Marahleh, Yasuhiko Nara, Ria Kinjo, Jinghan Ma, Kayoko Kanou, Yukinori Tanaka, Itaru Mizoguchi

C‑X‑C receptor 7 agonist acts as a C‑X‑C motif chemokine ligand 12 inhibitor to ameliorate osteoclastogenesis and bone resorption

C‑X‑C 受体 7 激动剂可作为 C‑X‑C 基序趋化因子配体 12 抑制剂，改善破骨细胞生成和骨吸收

期刊:Molecular Medicine Reports

影响因子:3.4

doi:10.3892/mmr.2022.12594

Alexander Patera Nugraha #, Hideki Kitaura, Fumitoshi Ohori #, Adya Pramusita, Saika Ogawa, Takahiro Noguchi, Aseel Marahleh, Yasuhiko Nara, Ria Kinjo, Itaru Mizoguchi

Inhibition of the CXCL9-CXCR3 axis suppresses the progression of experimental apical periodontitis by blocking macrophage migration and activation

抑制 CXCL9-CXCR3 轴可通过阻断巨噬细胞迁移和活化来抑制实验性根尖牙周炎的进展

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-021-82167-7

Tatsuya Hasegawa, V Venkata Suresh, Yoshio Yahata, Masato Nakano, Shigeto Suzuki, Shigeki Suzuki, Satoru Yamada, Hideki Kitaura, Itaru Mizoguchi, Yuichiro Noiri, Keisuke Handa, Masahiro Saito

Angiotensin II Promotes Osteocyte RANKL Expression via AT1R Activation

血管紧张素 II 通过激活 AT1R 促进骨细胞 RANKL 表达

Role of CXCL10 released from osteocytes in response to TNF-α stimulation on osteoclasts

CXCL10在骨细胞响应TNF-α刺激对破骨细胞的作用

Exogenous Angiotensin-(1-7) Provides Protection Against Inflammatory Bone Resorption and Osteoclastogenesis by Inhibition of TNF-α Expression in Macrophages

外源性血管紧张素-(1-7) 通过抑制巨噬细胞中的 TNF-α 表达来防止炎症性骨吸收和破骨细胞生成

(D-Ala2)GIP Inhibits Inflammatory Bone Resorption by Suppressing TNF-α and RANKL Expression and Directly Impeding Osteoclast Formation

(D-Ala2)GIP 通过抑制 TNF-α 和 RANKL 表达并直接阻碍破骨细胞形成来抑制炎症性骨吸收

Fermented Rice Bran Supplementation Inhibits LPS-Induced Osteoclast Formation and Bone Resorption in Mice

发酵米糠补充剂可抑制小鼠 LPS 诱导的破骨细胞形成和骨吸收

Generating Bone Marrow Chimeric Mouse Using GPR120 Deficient Mouse for the Study of DHA Inhibitory Effect on Osteoclast Formation and Bone Resorption

利用GPR120缺陷小鼠制备骨髓嵌合小鼠，研究DHA对破骨细胞形成和骨吸收的抑制作用

Azilsartan inhibits inflammation-triggered bone resorption and osteoclastogenesis in vivo via suppression of TNF-α expression in macrophages

阿齐沙坦通过抑制巨噬细胞中的 TNF-α 表达来抑制体内炎症引发的骨吸收和破骨细胞生成

Salt-Sensitive Hypertension Induces Osteoclastogenesis and Bone Resorption via Upregulation of Angiotensin II Type 1 Receptor Expression in Osteoblasts

盐敏感性高血压通过上调成骨细胞中血管紧张素 II 1 型受体的表达来诱导破骨细胞生成和骨吸收

C‑X‑C receptor 7 agonist acts as a C‑X‑C motif chemokine ligand 12 inhibitor to ameliorate osteoclastogenesis and bone resorption

C‑X‑C 受体 7 激动剂可作为 C‑X‑C 基序趋化因子配体 12 抑制剂，改善破骨细胞生成和骨吸收

Inhibition of the CXCL9-CXCR3 axis suppresses the progression of experimental apical periodontitis by blocking macrophage migration and activation

抑制 CXCL9-CXCR3 轴可通过阻断巨噬细胞迁移和活化来抑制实验性根尖牙周炎的进展