Jamie C Zampell相关文献与解析，生知库 | 链接生命科学的每一步

Swapna Ghanta, Daniel A Cuzzone, Jeremy S Torrisi, Nicholas J Albano, Walter J Joseph, Ira L Savetsky, Jason C Gardenier, David Chang, Jamie C Zampell, Babak J Mehrara

免疫

细胞生物学

巨噬细胞

Immunology/Inflammation

发表日期：2015

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Th2 differentiation is necessary for soft tissue fibrosis and lymphatic dysfunction resulting from lymphedema

Th2 分化是淋巴水肿导致的软组织纤维化和淋巴功能障碍所必需的

期刊:FASEB Journal

影响因子:4.4

doi:10.1096/fj.12-222695

Tomer Avraham, Jamie C Zampell, Alan Yan, Sonia Elhadad, Evan S Weitman, Stanley G Rockson, Jacqueline Bromberg, Babak J Mehrara

细胞生物学

发表日期：2013

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Obesity impairs lymphatic fluid transport and dendritic cell migration to lymph nodes

肥胖会损害淋巴液运输和树突状细胞向淋巴结的迁移

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0070703

Evan S Weitman, Seth Z Aschen, Gina Farias-Eisner, Nicholas Albano, Daniel A Cuzzone, Swapna Ghanta, Jamie C Zampell, Daniel Thorek, Babak J Mehrara

HIF-1α coordinates lymphangiogenesis during wound healing and in response to inflammation

HIF-1α 在伤口愈合和炎症反应过程中协调淋巴管生成

期刊:FASEB Journal

影响因子:4.4

doi:10.1096/fj.11-195321

Jamie C Zampell, Alan Yan, Tomer Avraham, Sanjay Daluvoy, Evan S Weitman, Babak J Mehrara

Regulation of adipogenesis by lymphatic fluid stasis: part I. Adipogenesis, fibrosis, and inflammation

淋巴液淤滞对脂肪生成的调节：第一部分脂肪生成、纤维化和炎症

期刊:Plastic and Reconstructive Surgery

影响因子:3.2

doi:10.1097/PRS.0b013e3182450b2d

Jamie C Zampell, Seth Aschen, Evan S Weitman, Alan Yan, Sonia Elhadad, Marina De Brot Andrade, Babak J Mehrara

信号转导

发表日期：2012

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Temporal and spatial patterns of endogenous danger signal expression after wound healing and in response to lymphedema

伤口愈合后和淋巴水肿反应中内源性危险信号表达的时间和空间模式

期刊:American Journal of Physiology-Cell Physiology

影响因子:5

doi:10.1152/ajpcell.00378.2010

Jamie C Zampell, Alan Yan, Tomer Avraham, Victor Andrade, Stephanie Malliaris, Seth Aschen, Stanley G Rockson, Babak J Mehrara

信号转导

发表日期：2011

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Adipose-derived stem cells promote lymphangiogenesis in response to VEGF-C stimulation or TGF-β1 inhibition

脂肪干细胞在 VEGF-C 刺激或 TGF-β1 抑制下促进淋巴管生成

期刊:Future Oncology

影响因子:3

doi:10.2217/fon.11.121

Alan Yan, Tomer Avraham, Jamie C Zampell, Yosef S Haviv, Evan Weitman, Babak J Mehrara

Regulation of inflammation and fibrosis by macrophages in lymphedema

巨噬细胞对淋巴水肿炎症和纤维化的调节

Th2 differentiation is necessary for soft tissue fibrosis and lymphatic dysfunction resulting from lymphedema

Th2 分化是淋巴水肿导致的软组织纤维化和淋巴功能障碍所必需的

Obesity impairs lymphatic fluid transport and dendritic cell migration to lymph nodes

肥胖会损害淋巴液运输和树突状细胞向淋巴结的迁移

HIF-1α coordinates lymphangiogenesis during wound healing and in response to inflammation

HIF-1α 在伤口愈合和炎症反应过程中协调淋巴管生成

Regulation of adipogenesis by lymphatic fluid stasis: part I. Adipogenesis, fibrosis, and inflammation

淋巴液淤滞对脂肪生成的调节：第一部分脂肪生成、纤维化和炎症

Temporal and spatial patterns of endogenous danger signal expression after wound healing and in response to lymphedema

伤口愈合后和淋巴水肿反应中内源性危险信号表达的时间和空间模式

Adipose-derived stem cells promote lymphangiogenesis in response to VEGF-C stimulation or TGF-β1 inhibition

脂肪干细胞在 VEGF-C 刺激或 TGF-β1 抑制下促进淋巴管生成