日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Molecular structural diversity of mitochondrial cardiolipins

线粒体心磷脂的分子结构多样性

Oemer, Gregor; Lackner, Katharina; Muigg, Katharina; Krumschnabel, Gerhard; Watschinger, Katrin; Sailer, Sabrina; Lindner, Herbert; Gnaiger, Erich; Wortmann, Saskia B; Werner, Ernst R; Zschocke, Johannes; Keller, Markus A

The cyanobacterial metabolite nocuolin a is a natural oxadiazine that triggers apoptosis in human cancer cells

蓝藻代谢物 nocuolin a 是一种天然的恶二嗪,可引发人类癌细胞凋亡

Kateřina Voráčová, Jan Hájek, Jan Mareš, Petra Urajová, Marek Kuzma, José Cheel, Andreas Villunger, Alexandra Kapuscik, Marcel Bally, Petr Novák, Martin Kabeláč, Gerhard Krumschnabel, Martin Lukeš, Ludmila Voloshko, Jiří Kopecký, Pavel Hrouzek

Mis-targeting of the mitochondrial protein LIPT2 leads to apoptotic cell death

线粒体蛋白 LIPT2 的错误靶向导致细胞凋亡

Emanuele Bernardinelli, Roberta Costa, Giada Scantamburlo, Janet To, Rossana Morabito, Charity Nofziger, Carolina Doerrier, Gerhard Krumschnabel, Markus Paulmichl, Silvia Dossena

High-Resolution Respirometry for Simultaneous Measurement of Oxygen and Hydrogen Peroxide Fluxes in Permeabilized Cells, Tissue Homogenate and Isolated Mitochondria

高分辨率呼吸测量法可同时测量透化细胞、组织匀浆和分离线粒体中的氧气和过氧化氢通量

Makrecka-Kuka, Marina; Krumschnabel, Gerhard; Gnaiger, Erich

The p53 binding protein PDCD5 is not rate-limiting in DNA damage induced cell death

p53 结合蛋白 PDCD5 不是 DNA 损伤诱导细胞死亡的限速因素

Florian J Bock, Maria C Tanzer, Manuel D Haschka, Gerhard Krumschnabel, Bénédicte Sohm, Katrin Goetsch, Reinhard Kofler, Andreas Villunger

Loss of PIDD limits NF-κB activation and cytokine production but not cell survival or transformation after DNA damage

PIDD的缺失会限制NF-κB的激活和细胞因子的产生,但不会影响DNA损伤后的细胞存活或转化。

F J Bock ,G Krumschnabel, C Manzl, L Peintner, M C Tanzer, N Hermann-Kleiter, G Baier, L Llacuna, J Yelamos, A Villunger

Death of p53-defective cells triggered by forced mitotic entry in the presence of DNA damage is not uniquely dependent on Caspase-2 or the PIDDosome

在 DNA 损伤的情况下,强制有丝分裂进入引发的 p53 缺陷细胞死亡并非仅仅依赖于 Caspase-2 或 PIDDosome

C Manzl, L L Fava, G Krumschnabel, L Peintner, M C Tanzer, C Soratroi, F J Bock, F Schuler, B Luef, S Geley, A Villunger

PIDDosome-independent tumor suppression by Caspase-2

Caspase-2 实现 PIDDosome 独立的肿瘤抑制

C Manzl, L Peintner, G Krumschnabel, F Bock, V Labi, M Drach, A Newbold, R Johnstone, Andreas Villunger

Necrosis-like death can engage multiple pro-apoptotic Bcl-2 protein family members

坏死样死亡可能涉及多个促凋亡的 Bcl-2 蛋白家族成员

Denise Tischner #, Claudia Manzl #, Claudia Soratroi, Andreas Villunger, Gerhard Krumschnabel

BH3-only protein Bmf mediates apoptosis upon inhibition of CAP-dependent protein synthesis

BH3 特异性蛋白 Bmf 在抑制 CAP 依赖性蛋白质合成后介导细胞凋亡

F Grespi, C Soratroi, G Krumschnabel, B Sohm, C Ploner, S Geley, L Hengst, G Häcker, A Villunger