Lucio G相关文献与解析，生知库 | 链接生命科学的每一步

Do varsity college athletes have a greater likelihood of risky alcohol and cannabis use than non-athletes? Results from a National Survey in Brazil

大学运动员比非运动员更容易出现危险的酒精和大麻使用行为吗？巴西一项全国性调查的结果

doi:10.1590/1516-4446-2021-2236

Mannes, Zachary L; Hasin, Deborah S; Martins, Silvia S; Gonçalves, Priscila D; Livne, Ofir; de Oliveira, Lucio G; de Andrade, Arthur G; McReynolds, Larkin S; McDuff, David; Hainline, Brian; Castaldelli-Maia, João M

发表日期：2022

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Examining the role of paraoxonase 2 in the dopaminergic system of the mouse brain

研究对氧磷酶 2 在小鼠大脑多巴胺系统中的作用

期刊:BMC Neuroscience

影响因子:2.4

doi:10.1186/s12868-022-00738-4

Jacqueline M Garrick, Khoi Dao, Lucio G Costa, Judit Marsillach, Clement E Furlong

Paraoxonase 2 deficiency in mice alters motor behavior and causes region-specific transcript changes in the brain

小鼠对氧磷酶2缺乏会改变其运动行为，并导致大脑特定区域的转录本发生改变。

期刊:Neurotoxicology and Teratology

影响因子:2.8

doi:10.1016/j.ntt.2021.107010

Garrick, Jacqueline M; Cole, Toby B; Bammler, Theo K; MacDonald, James W; Marsillach, Judit; Furlong, Clement E; Costa, Lucio G

神经科学

发表日期：2021

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Evaluating Gait and Locomotion in Rodents with the CatWalk

利用CatWalk评估啮齿动物的步态和运动能力

期刊:Current Protocols

影响因子:2.2

doi:10.1002/cpz1.220

Garrick, Jacqueline M; Costa, Lucio G; Cole, Toby B; Marsillach, Judit

发表日期：2021

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Paraoxonase-1 (PON1) Status Analysis Using Non-Organophosphate Substrates.

利用非有机磷酸酯底物进行对氧磷酶-1 (PON1) 状态分析

期刊:Current Protocols

影响因子:2.2

doi:10.1002/cpz1.25

Marsillach Judit, Richter Rebecca J, Costa Lucio G, Furlong Clement E

表观遗传

发表日期：2021

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Effects of air pollution on the nervous system and its possible role in neurodevelopmental and neurodegenerative disorders

空气污染对神经系统的影响及其在神经发育和神经退行性疾病中的可能作用

期刊:Pharmacology & Therapeutics

影响因子:12.5

doi:10.1016/j.pharmthera.2020.107523

Costa, Lucio G; Cole, Toby B; Dao, Khoi; Chang, Yu-Chi; Coburn, Jacki; Garrick, Jacqueline M

Developmental exposure to diesel exhaust upregulates transcription factor expression, decreases hippocampal neurogenesis, and alters cortical lamina organization: relevance to neurodevelopmental disorders.

发育过程中暴露于柴油尾气会促进转录因子表达，降低海马神经发生，并改变皮质层组织：与神经发育障碍相关

期刊:Journal of Neurodevelopmental Disorders

影响因子:4

doi:10.1186/s11689-020-09340-3

Cole Toby B, Chang Yu-Chi, Dao Khoi, Daza Ray, Hevner Robert, Costa Lucio G

神经科学

发表日期：2020

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Prenatal and early life diesel exhaust exposure disrupts cortical lamina organization: Evidence for a reelin-related pathogenic pathway induced by interleukin-6

胎儿期和生命早期柴油废气暴露会破坏皮质层组织：白细胞介素-6 诱导的 reelin 相关致病途径的证据

期刊:Brain Behavior and Immunity

影响因子:8.8

doi:10.1016/j.bbi.2019.01.013

Yu-Chi Chang, Ray Daza, Robert Hevner, Lucio G Costa, Toby B Cole

Synaptogenesis by Cholinergic Stimulation of Astrocytes

胆碱能刺激星形胶质细胞促进突触形成

Perspective on halogenated organic compounds

卤代有机化合物的视角

Do varsity college athletes have a greater likelihood of risky alcohol and cannabis use than non-athletes? Results from a National Survey in Brazil

大学运动员比非运动员更容易出现危险的酒精和大麻使用行为吗？巴西一项全国性调查的结果

Examining the role of paraoxonase 2 in the dopaminergic system of the mouse brain

研究对氧磷酶 2 在小鼠大脑多巴胺系统中的作用

Paraoxonase 2 deficiency in mice alters motor behavior and causes region-specific transcript changes in the brain

小鼠对氧磷酶2缺乏会改变其运动行为，并导致大脑特定区域的转录本发生改变。

Evaluating Gait and Locomotion in Rodents with the CatWalk

利用CatWalk评估啮齿动物的步态和运动能力

Paraoxonase-1 (PON1) Status Analysis Using Non-Organophosphate Substrates.

利用非有机磷酸酯底物进行对氧磷酶-1 (PON1) 状态分析

Effects of air pollution on the nervous system and its possible role in neurodevelopmental and neurodegenerative disorders

空气污染对神经系统的影响及其在神经发育和神经退行性疾病中的可能作用

Developmental exposure to diesel exhaust upregulates transcription factor expression, decreases hippocampal neurogenesis, and alters cortical lamina organization: relevance to neurodevelopmental disorders.

发育过程中暴露于柴油尾气会促进转录因子表达，降低海马神经发生，并改变皮质层组织：与神经发育障碍相关

Prenatal and early life diesel exhaust exposure disrupts cortical lamina organization: Evidence for a reelin-related pathogenic pathway induced by interleukin-6

胎儿期和生命早期柴油废气暴露会破坏皮质层组织：白细胞介素-6 诱导的 reelin 相关致病途径的证据