日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Design and advanced characterization of quercetin-loaded nano-liposomes prepared by high-pressure homogenization

高压均质法制备槲皮素纳米脂质体的设计和高级表征

Sofia Melchior, Marta Codrich, Andrea Gorassini, Dora Mehn, Jessica Ponti, Giancarlo Verardo, Gianluca Tell, Luigi Calzolai, Sonia Calligaris

AUF1 Recognizes 8-Oxo-Guanosine Embedded in DNA and Stimulates APE1 Endoribonuclease Activity

AUF1 识别 DNA 中嵌入的 8-氧代鸟苷并刺激 APE1 内切核糖核酸酶活性

Matilde Clarissa Malfatti, Marta Codrich, Emiliano Dalla, Chiara D'Ambrosio, Francesca Storici, Andrea Scaloni, Gianluca Tell

APE1 interacts with the nuclear exosome complex protein MTR4 and is involved in cisplatin- and 5-fluorouracil-induced RNA damage response

APE1 与核外泌体复合蛋白 MTR4 相互作用,参与顺铂和 5-氟尿嘧啶诱导的 RNA 损伤反应

Marta Codrich, Monica Degrassi, Matilde Clarissa Malfatti, Giulia Antoniali, Andrea Gorassini, Dilara Ayyildiz, Rossella De Marco, Giancarlo Verardo, Gianluca Tell

Neuronal haemoglobin induces loss of dopaminergic neurons in mouse Substantia nigra, cognitive deficits and cleavage of endogenous α-synuclein

神经元血红蛋白诱导小鼠黑质多巴胺能神经元丢失、认知缺陷和内源性 α-突触核蛋白裂解

Chiara Santulli #, Carlotta Bon #, Elena De Cecco, Marta Codrich, Joanna Narkiewicz, Pietro Parisse, Fabio Perissinotto, Claudio Santoro, Francesca Persichetti, Giuseppe Legname, Stefano Espinoza, Stefano Gustincich

Inhibition of APE1-endonuclease activity affects cell metabolism in colon cancer cells via a p53-dependent pathway

抑制 APE1 内切酶活性通过 p53 依赖性途径影响结肠癌细胞的细胞代谢

Marta Codrich, Marina Comelli, Matilde Clarissa Malfatti, Catia Mio, Dilara Ayyildiz, Chi Zhang, Mark R Kelley, Giovanni Terrosu, Carlo E M Pucillo, Gianluca Tell

Neuronal hemoglobin affects dopaminergic cells' response to stress

神经元血红蛋白影响多巴胺能细胞对压力的反应

Marta Codrich, Maria Bertuzzi, Roberta Russo, Margherita Francescatto, Stefano Espinoza, Lorena Zentilin, Mauro Giacca, Daniela Cesselli, Antonio Paolo Beltrami, Paolo Ascenzi, Silvia Zucchelli, Francesca Persichetti, Giampiero Leanza, Stefano Gustincich

Parkinson's disease DJ-1 L166P alters rRNA biogenesis by exclusion of TTRAP from the nucleolus and sequestration into cytoplasmic aggregates via TRAF6

帕金森病 DJ-1 L166P 通过将 TTRAP 从核仁中排除并通过 TRAF6 隔离到细胞质聚集体中来改变 rRNA 的生物合成

Sandra Vilotti, Marta Codrich, Marco Dal Ferro, Milena Pinto, Isidro Ferrer, Licio Collavin, Stefano Gustincich, Silvia Zucchelli

Tumor necrosis factor receptor-associated factor 6 (TRAF6) associates with huntingtin protein and promotes its atypical ubiquitination to enhance aggregate formation

肿瘤坏死因子受体相关因子 6 (TRAF6) 与亨廷顿蛋白相关并促进其非典型泛素化以增强聚集体形成

Silvia Zucchelli, Federica Marcuzzi, Marta Codrich, Elena Agostoni, Sandra Vilotti, Marta Biagioli, Milena Pinto, Alisia Carnemolla, Claudio Santoro, Stefano Gustincich, Francesca Persichetti

TRAF6 promotes atypical ubiquitination of mutant DJ-1 and alpha-synuclein and is localized to Lewy bodies in sporadic Parkinson's disease brains

TRAF6 促进突变型 DJ-1 和 α-突触核蛋白的非典型泛素化,并定位于散发性帕金森病患者大脑中的路易体

Silvia Zucchelli, Marta Codrich, Federica Marcuzzi, Milena Pinto, Sandra Vilotti, Marta Biagioli, Isidro Ferrer, Stefano Gustincich