日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvironment

KRASG12R突变型胰腺癌具有ERK/MAPK转录活性受限和独特的肿瘤微环境。

Burge, Rachel A; Le Roux, Ozgun; Popow, Olesja; Spadafora, Victoria K; Rajesh, Christabelle; Adair, Sara J; Bialousow, Lucas; Murphy, Cailey; Saberi, Samaneh; Vaena, Silvia G; Taquey, Margaret C; Allen, Sarah; Han, Lu; Helke, Kristi L; Sharma, Sudarshana; Ostrowski, Michael C; Guttridge, Denis C; Dincman, Toros A; Bauer, Todd W; Kashatus, David F; McFall, Thomas; Haigis, Kevin M; Hollingsworth, Michael A; Counter, Christopher M; Der, Channing J; Hobbs, G Aaron

Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma.

突变型和野生型 RAS 串扰和化学计量缺陷是 KRASG12R 胰腺导管腺癌对靶向治疗敏感性的决定因素。

Kamgar Mandana, Hobbes G Aaron, Davidson Raven, Dorbin Daniel, Herrera Juliannie, Langenheim John F, Burge Rachel A, Aldakkak Mohammed, Conrardy Ryan D, Szabo Aniko, Thalji Sam Z, Mayer Bradley, Grahl Justin J, Chung Brian Y, Phan Alexandria, Thomas James P, Seo Y David, Evans Douglas B, Christians Kathleen K, Erickson Beth, Hall William A, George Ben, Tsai Susan, Lytle Nikki K, Der Channing J, Kurzrock Razelle, McFall Thomas

Mathematical modeling suggests 14-3-3 proteins modulate RAF paradoxical activation.

数学模型表明 14-3-3 蛋白调节 RAF 的矛盾激活

Mendiratta Gaurav, Abbott Kodye, Li Yao-Cheng, Yu Jingting, Carlip Peter, Tong Melinda, Huang Jianfeng, Shokhirev Maxim N, McFall Thomas, Wahl Geoffrey M, Stites Edward C

Pancreatic Cells Are Resistant to KRASQ61L Expression due to Hyperactive ERK/MAPK Signaling and Apoptosis Induction

胰腺细胞由于ERK/MAPK信号通路过度活跃和细胞凋亡诱导而对KRASQ61L表达具有抵抗性。

Burge, Rachel A; Bialousow, Lucas; McFall, Thomas; Bamonte, Logan; Johnson, Grayson; Smith, Merissa; Vaena, Silvia G; Comte-Walters, Susana; Ball, Lauren E; Berto, Stefano; O'Bryan, John P; Hobbs, G Aaron

Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, a case series

针对KRAS野生型胰腺导管腺癌融合基因和驱动基因突变的无化疗治疗:病例系列研究

Mehdi, Maahum; Szabo, Aniko; Shreenivas, Aditya; Thomas, James P; Tsai, Susan; Christians, Kathleen K; Evans, Douglas B; Clarke, Callisia N; Hall, William A; Erickson, Beth; Ahmed, Gulrayz; Thapa, Bicky; McFall, Thomas; George, Ben; Kurzrock, Razelle; Kamgar, Mandana

KRAS-Mutated, Estrogen Receptor-Positive Low-Grade Serous Ovarian Cancer: Unraveling an Exceptional Response Mystery

KRAS突变、雌激素受体阳性的低级别浆液性卵巢癌:揭开一种特殊疗效之谜

Kato, Shumei; McFall, Thomas; Takahashi, Kenta; Bamel, Kasey; Ikeda, Sadakatsu; Eskander, Ramez N; Plaxe, Steven; Parker, Barbara; Stites, Edward; Kurzrock, Razelle

A mechanism for the response of KRAS(G13D) expressing colorectal cancers to EGFR inhibitors

KRAS(G13D)表达型结直肠癌对EGFR抑制剂的反应机制

McFall, Thomas; Stites, Edward Cooper