Morgan N G相关文献与解析，生知库 | 链接生命科学的每一步

Inhalation of glutamic acid decarboxylase 65-derived peptides can protect against recurrent autoimmune but not alloimmune responses in the non-obese diabetic mouse

吸入谷氨酸脱羧酶65衍生的肽可以保护非肥胖糖尿病小鼠免受复发性自身免疫反应的侵害，但不能保护其免受同种异体免疫反应的侵害。

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1111/j.1365-2249.2007.03358.x

Ravanan, R; Wong, S F; Morgan, N G; Mathieson, P W; Smith, R M

Pre-incubation with interleukin-4 mediates a direct protective effect against the loss of pancreatic beta-cell viability induced by proinflammatory cytokines

用白细胞介素-4预孵育可直接保护胰岛β细胞免受促炎细胞因子诱导的细胞活力丧失。

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1111/j.1365-2249.2007.03375.x

Kaminski, A; Kaminski, E R; Morgan, N G

Islet glutamic acid decarboxylase modified by reactive oxygen species is recognized by antibodies from patients with type 1 diabetes mellitus.

1 型糖尿病患者的抗体可以识别被活性氧修饰的胰岛谷氨酸脱羧酶

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.2001.01653.x

Trigwell S M, Radford P M, Page S R, Loweth A C, James R F, Morgan N G, Todd I

The effect of the putative endogenous imidazoline receptor ligand, clonidine-displacing substance, on insulin secretion from rat and human islets of Langerhans

假定的内源性咪唑啉受体配体——可乐定置换物质——对大鼠和人胰岛胰岛素分泌的影响

期刊:British Journal of Pharmacology

影响因子:7.7

doi:10.1038/sj.bjp.0700964

Chan, S L; Atlas, D; James, R F; Morgan, N G

Interactions between imidazoline compounds and sulphonylureas in the regulation of insulin secretion

咪唑啉类化合物与磺酰脲类药物在胰岛素分泌调节中的相互作用

期刊:British Journal of Pharmacology

影响因子:7.7

doi:10.1038/sj.bjp.0701172

Mourtada, M; Brown, C A; Smith, S A; Piercy, V; Chan, S L; Morgan, N G

免疫/内分泌

发表日期：1997

文献求助收藏

Activation of alpha-2-adrenoceptors results in an increase in F-actin formation in HIT-T15 pancreatic B-cells

α2-肾上腺素受体的激活导致HIT-T15胰岛β细胞中F-肌动蛋白的形成增加。

期刊:Biochemical Journal

影响因子:4.3

doi:10.1042/bj3070169

Cable, H C; el-Mansoury, A; Morgan, N G

The imidazoline site involved in control of insulin secretion: characteristics that distinguish it from I1- and I2-sites

参与胰岛素分泌调控的咪唑啉位点：区别于 I1 位点和 I2 位点的特征

期刊:British Journal of Pharmacology

影响因子:7.7

doi:10.1111/j.1476-5381.1994.tb13191.x

Chan, S L; Brown, C A; Scarpello, K E; Morgan, N G

Antagonism of the stimulatory effects of efaroxan and glibenclamide in rat pancreatic islets by the imidazoline, RX801080

咪唑啉类化合物RX801080对依法罗沙和格列本脲在大鼠胰岛中的刺激作用的拮抗作用

期刊:British Journal of Pharmacology

影响因子:7.7

doi:10.1111/j.1476-5381.1993.tb13915.x

Brown, C A; Chan, S L; Stillings, M R; Smith, S A; Morgan, N G

Rat

发表日期：1993

文献求助收藏

Stimulation of insulin secretion by imidazoline compounds is not due to interaction with non-adrenoceptor idazoxan binding sites

咪唑啉类化合物刺激胰岛素分泌并非由于与非肾上腺素受体咪唑啉结合位点相互作用所致。

期刊:British Journal of Pharmacology

影响因子:7.7

doi:10.1111/j.1476-5381.1993.tb12801.x

Brown, C A; Loweth, A C; Smith, S A; Morgan, N G

Analysis of islet inflammation in human type 1 diabetes

人类1型糖尿病胰岛炎症分析

Inhalation of glutamic acid decarboxylase 65-derived peptides can protect against recurrent autoimmune but not alloimmune responses in the non-obese diabetic mouse

吸入谷氨酸脱羧酶65衍生的肽可以保护非肥胖糖尿病小鼠免受复发性自身免疫反应的侵害，但不能保护其免受同种异体免疫反应的侵害。

Pre-incubation with interleukin-4 mediates a direct protective effect against the loss of pancreatic beta-cell viability induced by proinflammatory cytokines

用白细胞介素-4预孵育可直接保护胰岛β细胞免受促炎细胞因子诱导的细胞活力丧失。

Islet glutamic acid decarboxylase modified by reactive oxygen species is recognized by antibodies from patients with type 1 diabetes mellitus.

1 型糖尿病患者的抗体可以识别被活性氧修饰的胰岛谷氨酸脱羧酶

The effect of the putative endogenous imidazoline receptor ligand, clonidine-displacing substance, on insulin secretion from rat and human islets of Langerhans

假定的内源性咪唑啉受体配体——可乐定置换物质——对大鼠和人胰岛胰岛素分泌的影响

Interactions between imidazoline compounds and sulphonylureas in the regulation of insulin secretion

咪唑啉类化合物与磺酰脲类药物在胰岛素分泌调节中的相互作用

Activation of alpha-2-adrenoceptors results in an increase in F-actin formation in HIT-T15 pancreatic B-cells

α2-肾上腺素受体的激活导致HIT-T15胰岛β细胞中F-肌动蛋白的形成增加。

The imidazoline site involved in control of insulin secretion: characteristics that distinguish it from I1- and I2-sites

参与胰岛素分泌调控的咪唑啉位点：区别于 I1 位点和 I2 位点的特征

Antagonism of the stimulatory effects of efaroxan and glibenclamide in rat pancreatic islets by the imidazoline, RX801080

咪唑啉类化合物RX801080对依法罗沙和格列本脲在大鼠胰岛中的刺激作用的拮抗作用

Stimulation of insulin secretion by imidazoline compounds is not due to interaction with non-adrenoceptor idazoxan binding sites

咪唑啉类化合物刺激胰岛素分泌并非由于与非肾上腺素受体咪唑啉结合位点相互作用所致。