Neng-Ming Lin相关文献与解析，生知库 | 链接生命科学的每一步

DYRK1A reinforces epithelial-mesenchymal transition and metastasis of hepatocellular carcinoma via cooperatively activating STAT3 and SMAD

DYRK1A 通过协同激活 STAT3 和 SMAD 增强肝细胞癌的上皮-间质转化和转移

期刊:Journal of Biomedical Science

影响因子:9

doi:10.1186/s12929-022-00817-y

Yang-Ling Li #, Man-Man Zhang #, Lin-Wen Wu, Ye-Han Liu, Zuo-Yan Zhang, Ling-Hui Zeng, Neng-Ming Lin, Chong Zhang

Decreased INPP5B expression predicts poor prognosis in lung adenocarcinoma

INPP5B 表达降低预示肺腺癌预后不良

期刊:Cancer Cell International

影响因子:5.3

doi:10.1186/s12935-022-02609-8

Jun Deng #, Xu Lin #, Qi Li, Xiao-Yu Cai, Lin-Wen Wu, Wei Wang, Bo Zhang, Yang-Ling Li, Jian Hu, Neng-Ming Lin

AKR1C1 promotes non-small cell lung cancer proliferation via crosstalk between HIF-1α and metabolic reprogramming

AKR1C1 通过 HIF-1α 与代谢重编程之间的串扰促进非小细胞肺癌增殖

期刊:Translational Oncology

影响因子:4.5

doi:10.1016/j.tranon.2022.101421

Lin-Lin Chang, Pei-Hua Lu, Wei Yang, Yan Hu, Lin Zheng, Qiong Zhao, Neng-Ming Lin, Wen-Zhou Zhang

Inhibition of TRIM32 by ibr-7 treatment sensitizes pancreatic cancer cells to gemcitabine via mTOR/p70S6K pathway

ibr-7 治疗抑制 TRIM32 可使胰腺癌细胞通过 mTOR/p70S6K 通路对吉西他滨敏感

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.3

doi:10.1111/jcmm.17109

Bo Zhang, You-You Yan, Yang-Qin Gu, Fei Teng, Xu Lin, Xing-Lu Zhou, Jin-Xin Che, Xiao-Wu Dong, Li-Xin Zhou, Neng-Ming Lin

A novel derivative of valepotriate inhibits the PI3K/AKT pathway and causes Noxa-dependent apoptosis in human pancreatic cancer cells

缬氨酸三酸的新型衍生物抑制 PI3K/AKT 通路并导致人类胰腺癌细胞发生 Noxa 依赖性凋亡

期刊:Acta Pharmacologica Sinica

影响因子:6.9

doi:10.1038/s41401-019-0354-1

You-You Yan #, Ke-Yu Shi #, Fei Teng #, Jing Chen, Jin-Xin Che, Xiao-Wu Dong, Neng-Ming Lin, Bo Zhang

ApoC1 promotes the metastasis of clear cell renal cell carcinoma via activation of STAT3

ApoC1 通过激活 STAT3 促进透明细胞肾细胞癌的转移

期刊:Oncogene

影响因子:6.9

doi:10.1038/s41388-020-01428-3

Yang-Ling Li #, Lin-Wen Wu #, Ling-Hui Zeng, Zuo-Yan Zhang, Wei Wang, Chong Zhang, Neng-Ming Lin

DYRK1A inhibition suppresses STAT3/EGFR/Met signalling and sensitizes EGFR wild-type NSCLC cells to AZD9291

DYRK1A 抑制可抑制 STAT3/EGFR/Met 信号传导，并使 EGFR 野生型 NSCLC 细胞对 AZD9291 敏感

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.3

doi:10.1111/jcmm.14609

Yang-Ling Li, Ke Ding, Xiu Hu, Lin-Wen Wu, Dong-Mei Zhou, Ming-Jun Rao, Neng-Ming Lin, Chong Zhang

BAY 87-2243 sensitizes hepatocellular carcinoma Hep3B cells to histone deacetylase inhibitors treatment via GSK-3β activation

BAY 87-2243 通过激活 GSK-3β 增强肝细胞癌 Hep3B 细胞对组蛋白去乙酰化酶抑制剂治疗的敏感性

期刊:Experimental and Therapeutic Medicine

影响因子:2.4

doi:10.3892/etm.2019.7500

Yang-Ling Li, Ming-Jun Rao, Ning-Yu Zhang, Lin-Wen Wu, Neng-Ming Lin, Chong Zhang

Shikonin sensitizes wild‑type EGFR NSCLC cells to erlotinib and gefitinib therapy

紫草素使野生型 EGFR NSCLC 细胞对厄洛替尼和吉非替尼疗法敏感

期刊:Molecular Medicine Reports

影响因子:3.4

doi:10.3892/mmr.2018.9347

Yang-Ling Li, Xiu Hu, Qing-Yu Li, Fei Wang, Bo Zhang, Ke Ding, Bi-Qin Tan, Neng-Ming Lin, Chong Zhang

Vortioxetine exhibits anti-glioblastoma activity via the PI3K-Akt signaling pathway

沃替西汀通过 PI3K-Akt 信号通路发挥抗胶质母细胞瘤活性

DYRK1A reinforces epithelial-mesenchymal transition and metastasis of hepatocellular carcinoma via cooperatively activating STAT3 and SMAD

DYRK1A 通过协同激活 STAT3 和 SMAD 增强肝细胞癌的上皮-间质转化和转移

Decreased INPP5B expression predicts poor prognosis in lung adenocarcinoma

INPP5B 表达降低预示肺腺癌预后不良

AKR1C1 promotes non-small cell lung cancer proliferation via crosstalk between HIF-1α and metabolic reprogramming

AKR1C1 通过 HIF-1α 与代谢重编程之间的串扰促进非小细胞肺癌增殖

Inhibition of TRIM32 by ibr-7 treatment sensitizes pancreatic cancer cells to gemcitabine via mTOR/p70S6K pathway

ibr-7 治疗抑制 TRIM32 可使胰腺癌细胞通过 mTOR/p70S6K 通路对吉西他滨敏感

A novel derivative of valepotriate inhibits the PI3K/AKT pathway and causes Noxa-dependent apoptosis in human pancreatic cancer cells

缬氨酸三酸的新型衍生物抑制 PI3K/AKT 通路并导致人类胰腺癌细胞发生 Noxa 依赖性凋亡

ApoC1 promotes the metastasis of clear cell renal cell carcinoma via activation of STAT3

ApoC1 通过激活 STAT3 促进透明细胞肾细胞癌的转移

DYRK1A inhibition suppresses STAT3/EGFR/Met signalling and sensitizes EGFR wild-type NSCLC cells to AZD9291

DYRK1A 抑制可抑制 STAT3/EGFR/Met 信号传导，并使 EGFR 野生型 NSCLC 细胞对 AZD9291 敏感

BAY 87-2243 sensitizes hepatocellular carcinoma Hep3B cells to histone deacetylase inhibitors treatment via GSK-3β activation

BAY 87-2243 通过激活 GSK-3β 增强肝细胞癌 Hep3B 细胞对组蛋白去乙酰化酶抑制剂治疗的敏感性

Shikonin sensitizes wild‑type EGFR NSCLC cells to erlotinib and gefitinib therapy

紫草素使野生型 EGFR NSCLC 细胞对厄洛替尼和吉非替尼疗法敏感