日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

An LRP1-binding motif in cellular prion protein replicates cell-signaling activities of the full-length protein

细胞朊病毒蛋白中的 LRP1 结合基序复制全长蛋白的细胞信号传导活性

Elisabetta Mantuano, Carlotta Zampieri, Pardis Azmoon, Cory B Gunner, Kyle R Heye, Steven L Gonias

An antibody that targets cell-surface glucose-regulated protein-78 inhibits expression of inflammatory cytokines and plasminogen activator inhibitors by macrophages

针对细胞表面葡萄糖调节蛋白 78 的抗体可抑制巨噬细胞表达炎性细胞因子和纤溶酶原激活剂抑制剂

Cory B Gunner, Pardis Azmoon, Elisabetta Mantuano, Lipsa Das, Carlotta Zampieri, Salvatore V Pizzo, Steven L Gonias

CHMP2A regulates tumor sensitivity to natural killer cell-mediated cytotoxicity

CHMP2A 调节肿瘤对自然杀伤细胞介导的细胞毒性的敏感性

Davide Bernareggi # ,Qi Xie # ,Briana C Prager ,Jiyoung Yun ,Luisjesus S Cruz ,Timothy V Pham ,William Kim ,Xiqing Lee ,Michael Coffey ,Cristina Zalfa ,Pardis Azmoon ,Huang Zhu ,Pablo Tamayo ,Jeremy N Rich ,Dan S Kaufman

Cellular prion protein in human plasma-derived extracellular vesicles promotes neurite outgrowth via the NMDA receptor-LRP1 receptor system

人类血浆来源的细胞外囊泡中的细胞朊病毒蛋白通过 NMDA 受体-LRP1 受体系统促进神经突生长

Steven L Gonias, Michael A Banki, Pardis Azmoon, Haylie K Romero, Christina J Sigurdson, Elisabetta Mantuano, Wendy M Campana

The LRP1/CD91 ligands, tissue-type plasminogen activator, α2-macroglobulin, and soluble cellular prion protein have distinct co-receptor requirements for activation of cell-signaling

LRP1/CD91配体、组织型纤溶酶原激活剂、α2-巨球蛋白和可溶性细胞朊蛋白激活细胞信号传导需要不同的辅助受体。

Elisabetta Mantuano ,Pardis Azmoon ,Michael A Banki ,Cory B Gunner ,Steven L Gonias

A Soluble PrPC Derivative and Membrane-Anchored PrPC in Extracellular Vesicles Attenuate Innate Immunity by Engaging the NMDA-R/LRP1 Receptor Complex

细胞外囊泡中的可溶性 PrPC 衍生物和膜锚定的 PrPC 通过与 NMDA-R/LRP1 受体复合物结合来减弱先天免疫

Elisabetta Mantuano, Pardis Azmoon, Michael A Banki, Christina J Sigurdson, Wendy M Campana, Steven L Gonias

Enzymatically Inactive Tissue-Type Plasminogen Activator Reverses Disease Progression in the Dextran Sulfate Sodium Mouse Model of Inflammatory Bowel Disease

酶活性缺失的组织型纤溶酶原激活剂可逆转葡聚糖硫酸钠诱导的小鼠炎症性肠病模型中的疾病进展

Lipsa Das ,Michael A Banki ,Pardis Azmoon ,Donald Pizzo ,Steven L Gonias

Deletion of the Gene Encoding the NMDA Receptor GluN1 Subunit in Schwann Cells Causes Ultrastructural Changes in Remak Bundles and Hypersensitivity in Pain Processing

雪旺细胞中 NMDA 受体 GluN1 亚基编码基因的缺失导致 Remak 束超微结构变化和疼痛处理中的高敏感性

Coralie Brifault, Haylie Romero, Alicia Van-Enoo, Don Pizzo, Pardis Azmoon, HyoJun Kwon, Chanond Nasamran, Steven L Gonias, Wendy M Campana

A soluble derivative of PrPC activates cell-signaling and regulates cell physiology through LRP1 and the NMDA receptor

PrPC 的可溶性衍生物通过 LRP1 和 NMDA 受体激活细胞信号传导并调节细胞生理

Elisabetta Mantuano, Pardis Azmoon, Michael A Banki, Michael S Lam, Christina J Sigurdson, Steven L Gonias

Fibrinolysis protease receptors promote activation of astrocytes to express pro-inflammatory cytokines

纤维蛋白溶解蛋白酶受体促进星形胶质细胞活化并表达促炎细胞因子

Paola Pontecorvi, Michael A Banki, Carlotta Zampieri, Cristina Zalfa, Pardis Azmoon, Maria Z Kounnas, Cinzia Marchese, Steven L Gonias, Elisabetta Mantuano