日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Liquid Biopsy for Early Pancreatic Cancer Detection: Why Has It Not Yet Worked?

液体活检在胰腺癌早期检测中的应用:为何至今尚未取得成功?

Takahashi, Kenji; Ono, Yusuke; Taniue, Kenzui; Patra, Krushna C; Yamamoto, Takuya; Fujiya, Mikihiro; Mizukami, Yusuke

A Review of Multi-Agent AI Systems for Biological and Clinical Data Analysis

生物和临床数据分析的多智能体人工智能系统综述

Spieser, Jackson; Balapour, Ali; Meller, Jarek; Patra, Krushna C; Shamsaei, Behrouz

Correction: DNAJB1-PRKACA Fusion Drives Fibrolamellar Liver Cancer through Impaired SIK Signaling and CRTC2/p300-Mediated Transcriptional Reprogramming

更正:DNAJB1-PRKACA融合通过损害SIK信号传导和CRTC2/p300介导的转录重编程驱动纤维板层型肝癌

Gritti, Ilaria; Wan, Jinkai; Weeresekara, Vajira; Vaz, Joel M; Tarantino, Giuseppe; Bryde, Tenna Holgersen; Vijay, Vindhya; Kammula, Ashwin V; Kattel, Prabhat; Zhu, Songli; Vu, Phuong; Chan, Marina; Wu, Meng-Ju; Gordan, John D; Patra, Krushna C; Silveira, Vanessa S; Manguso, Robert T; Wein, Marc N; Ott, Christopher J; Qi, Jun; Liu, David; Sakamoto, Kei; Gujral, Taranjit S; Bardeesy, Nabeel

DNAJB1-PRKACA Fusion Drives Fibrolamellar Liver Cancer through Impaired SIK Signaling and CRTC2/p300-Mediated Transcriptional Reprogramming.

DNAJB1-PRKACA融合通过损害SIK信号传导和CRTC2/p300介导的转录重编程驱动纤维板层肝癌

Gritti Ilaria, Wan Jinkai, Weeresekara Vajira, Vaz Joel M, Tarantino Giuseppe, Bryde Tenna Holgersen, Vijay Vindhya, Kammula Ashwin V, Kattel Prabhat, Zhu Songli, Vu Phuong, Chan Marina, Wu Meng-Ju, Gordan John D, Patra Krushna C, Silveira Vanessa S, Manguso Robert T, Wein Marc N, Ott Christopher J, Qi Jun, Liu David, Sakamoto Kei, Gujral Taranjit S, Bardeesy Nabeel

Diversity of Precursor Lesions For Pancreatic Cancer: The Genetics and Biology of Intraductal Papillary Mucinous Neoplasm

胰腺癌前病变的多样性:导管内乳头状黏液性肿瘤的遗传学和生物学

Patra, Krushna C; Bardeesy, Nabeel; Mizukami, Yusuke

Hexokinase 2 as oncotarget

己糖激酶2作为癌靶点

Patra, Krushna C; Hay, Nissim

mTORC1 hyperactivity inhibits serum deprivation-induced apoptosis via increased hexokinase II and GLUT1 expression, sustained Mcl-1 expression, and glycogen synthase kinase 3beta inhibition

mTORC1 过度活跃通过增加己糖激酶 II 和 GLUT1 的表达、维持 Mcl-1 的表达以及抑制糖原合成酶激酶 3β 来抑制血清剥夺诱导的细胞凋亡。

Bhaskar, Prashanth T; Nogueira, Veronique; Patra, Krushna C; Jeon, Sang-Min; Park, Youngkyu; Robey, R Brooks; Hay, Nissim