Piedras-Rentería相关文献与解析，生知库

Genetic Deletion of KLHL1 Leads to Hyperexcitability in Hypothalamic POMC Neurons and Lack of Electrical Responses to Leptin

KLHL1 基因缺失导致下丘脑 POMC 神经元过度兴奋，缺乏对瘦素的电反应

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2021.718464

Paula P Perissinotti, Elizabeth Martínez-Hernández, Yungui He, Michael D Koob, Erika S Piedras-Rentería

神经

发表日期：2021

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KLHL1 Controls CaV3.2 Expression in DRG Neurons and Mechanical Sensitivity to Pain

KLHL1 控制 DRG 神经元中的 CaV3.2 表达和对疼痛的机械敏感性

期刊:Frontiers in Molecular Neuroscience

影响因子:3.5

doi:10.3389/fnmol.2019.00315

Elizabeth Martínez-Hernández, Alissa Zeglin, Erik Almazan, Paula Perissinotti, Yungui He, Michael Koob, Jody L Martin, Erika S Piedras-Rentería

神经

发表日期：2020

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Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice

Kelch-like 1 基因敲除小鼠海马神经元钙电流稳态和突触缺陷

期刊:Frontiers in Cellular Neuroscience

影响因子:4.2

doi:10.3389/fncel.2014.00444

Paula P Perissinotti, Elizabeth A Ethington, Erik Almazan, Elizabeth Martínez-Hernández, Jennifer Kalil, Michael D Koob, Erika S Piedras-Rentería

神经

发表日期：2015

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Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels

缺乏 P/Q 型 Ca2+ 通道的小鼠终板量子神经递质释放特性改变

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.0437991100

Francisco J Urbano, Erika S Piedras-Rentería, Kisun Jun, Hee-Sup Shin, Osvaldo D Uchitel, Richard W Tsien

神经

发表日期：2003

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Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit

缺乏α(1A)亚基的小鼠出现P/Q型Ca(2+)通道电流消失、突触传递改变和进行性共济失调。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.96.26.15245

Jun, K; Piedras-Rentería, E S; Smith, S M; Wheeler, D B; Lee, S B; Lee, T G; Chin, H; Adams, M E; Scheller, R H; Tsien, R W; Shin, H S

神经科学

发表日期：1999

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Antisense oligonucleotides against alpha1E reduce R-type calcium currents in cerebellar granule cells

针对α1E的反义寡核苷酸可降低小脑颗粒细胞中的R型钙电流。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.95.13.7760

Piedras-Rentería, E S; Tsien, R W

Antisense oligonucleotides against rat brain alpha1E DNA and its atrial homologue decrease T-type calcium current in atrial myocytes

针对大鼠脑α1E DNA及其心房同源物的反义寡核苷酸可降低心房肌细胞中的T型钙电流。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.94.26.14936

Piedras-Rentería, E S; Chen, C C; Best, P M

TRPC1/5-Ca V 3 Complex Mediates Leptin-Induced Excitability in Hypothalamic Neurons

TRPC1/5-Ca V3复合物介导瘦素诱导的下丘脑神经元兴奋性

Genetic Deletion of KLHL1 Leads to Hyperexcitability in Hypothalamic POMC Neurons and Lack of Electrical Responses to Leptin

KLHL1 基因缺失导致下丘脑 POMC 神经元过度兴奋，缺乏对瘦素的电反应

KLHL1 Controls CaV3.2 Expression in DRG Neurons and Mechanical Sensitivity to Pain

KLHL1 控制 DRG 神经元中的 CaV3.2 表达和对疼痛的机械敏感性

Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice

Kelch-like 1 基因敲除小鼠海马神经元钙电流稳态和突触缺陷

Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels

缺乏 P/Q 型 Ca2+ 通道的小鼠终板量子神经递质释放特性改变

Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit

缺乏α(1A)亚基的小鼠出现P/Q型Ca(2+)通道电流消失、突触传递改变和进行性共济失调。

Antisense oligonucleotides against alpha1E reduce R-type calcium currents in cerebellar granule cells

针对α1E的反义寡核苷酸可降低小脑颗粒细胞中的R型钙电流。

Antisense oligonucleotides against rat brain alpha1E DNA and its atrial homologue decrease T-type calcium current in atrial myocytes

针对大鼠脑α1E DNA及其心房同源物的反义寡核苷酸可降低心房肌细胞中的T型钙电流。