Pittala Srinivas相关文献与解析，生知库 | 链接生命科学的每一步

Mitochondrial VDAC1 Silencing in Urethane-Induced Lung Cancer Inhibits Tumor Growth and Alters Cancer Oncogenic Properties.

氨基甲酸乙酯诱导的肺癌中线粒体VDAC1沉默抑制肿瘤生长并改变癌症致癌特性

期刊:Cancers

影响因子:4.4

doi:10.3390/cancers16172970

Melnikov Nataly, Pittala Srinivas, Shteinfer-Kuzmine Anna, Shoshan-Barmatz Varda

Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

在阿尔茨海默病小鼠模型中，靶向过度表达的线粒体蛋白VDAC1可预防线粒体功能障碍并减轻脑部病理。

期刊:Translational Neurodegeneration

影响因子:15.2

doi:10.1186/s40035-022-00329-7

Verma, Ankit; Shteinfer-Kuzmine, Anna; Kamenetsky, Nikita; Pittala, Srinivas; Paul, Avijit; Nahon Crystal, Edna; Ouro, Alberto; Chalifa-Caspi, Vered; Pandey, Swaroop Kumar; Monsonego, Alon; Vardi, Noga; Knafo, Shira; Shoshan-Barmatz, Varda

The role of the mitochondrial protein VDAC1 in inflammatory bowel disease: a potential therapeutic target

线粒体蛋白VDAC1在炎症性肠病中的作用：潜在的治疗靶点

期刊:Molecular Therapy

影响因子:12

doi:10.1016/j.ymthe.2021.06.024

Verma, Ankit; Pittala, Srinivas; Alhozeel, Belal; Shteinfer-Kuzmine, Anna; Ohana, Ehud; Gupta, Rajeev; Chung, Jay H; Shoshan-Barmatz, Varda

A Mitochondrial VDAC1-Based Peptide Greatly Suppresses Steatosis and NASH-Associated Pathologies in a Mouse Model

一种基于线粒体VDAC1的肽在小鼠模型中显著抑制脂肪变性和NASH相关病理

期刊:Molecular Therapy

影响因子:12

doi:10.1016/j.ymthe.2019.06.017

Pittala, Srinivas; Krelin, Yakov; Kuperman, Yael; Shoshan-Barmatz, Varda

Mouse

发表日期：2019

文献求助收藏

Rewiring of Cancer Cell Metabolism by Mitochondrial VDAC1 Depletion Results in Time-Dependent Tumor Reprogramming: Glioblastoma as a Proof of Concept

线粒体VDAC1耗竭导致癌细胞代谢重编程，进而引发时间依赖性肿瘤重编程：以胶质母细胞瘤为例进行概念验证

期刊:Cells

影响因子:5.2

doi:10.3390/cells8111330

Arif, Tasleem; Stern, Oriel; Pittala, Srinivas; Chalifa-Caspi, Vered; Shoshan-Barmatz, Varda

Hepatic GRK2 is dispensable for glucose homeostasis and other key metabolic parameters in mice.

肝脏 GRK2 对于小鼠的葡萄糖稳态和其他关键代谢参数而言并非必需

Mitochondrial VDAC1 Silencing in Urethane-Induced Lung Cancer Inhibits Tumor Growth and Alters Cancer Oncogenic Properties.

氨基甲酸乙酯诱导的肺癌中线粒体VDAC1沉默抑制肿瘤生长并改变癌症致癌特性

Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

在阿尔茨海默病小鼠模型中，靶向过度表达的线粒体蛋白VDAC1可预防线粒体功能障碍并减轻脑部病理。

The role of the mitochondrial protein VDAC1 in inflammatory bowel disease: a potential therapeutic target

线粒体蛋白VDAC1在炎症性肠病中的作用：潜在的治疗靶点

A Mitochondrial VDAC1-Based Peptide Greatly Suppresses Steatosis and NASH-Associated Pathologies in a Mouse Model

一种基于线粒体VDAC1的肽在小鼠模型中显著抑制脂肪变性和NASH相关病理

Rewiring of Cancer Cell Metabolism by Mitochondrial VDAC1 Depletion Results in Time-Dependent Tumor Reprogramming: Glioblastoma as a Proof of Concept

线粒体VDAC1耗竭导致癌细胞代谢重编程，进而引发时间依赖性肿瘤重编程：以胶质母细胞瘤为例进行概念验证