日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Ceramide-rich extracellular vesicles as pathogenic biomarkers in traumatic brain injury

富含神经酰胺的细胞外囊泡作为创伤性脑损伤的致病生物标志物

Quadri, Zainuddin; Zhu, Zhihui; Ren, Xiaojia; Crivelli, Simone M; Zhang, Liping; Kunjadia, P D; Sullivan, Patrick G; Broome, Bibi B; Yamasaki, Tritia R; Bieberich, Erhard

Probing tau citrullination in Alzheimer's disease brains and mouse models of tauopathy.

探究阿尔茨海默病患者脑组织和 tau 蛋白病小鼠模型中的 tau 蛋白瓜氨酸化。

Liang Huimin, Hunt Jerry B Jr, Ma Chao, Kovalenko Andrii, Calahatian John, Pedersen Cecelie, Liu Haiying, Li Junyan, Serrano Malina, Blazier Danielle, Watler Mallory, Rocha-Rangel Patricia, Saunders Christopher, Blair Laura J, Breydo Leonid, Nash Kevin, Quadri Zainuddin, Kraemer Brian, Nelson Peter, Norris Christopher, Abner Erin L, Uversky Vladimir N, Chaput Dale, Selenica Maj-Linda B, Lee Daniel C

Staying sane in the membrane: Neutral sphingomyelinase 2 as a master regulator of plasma membrane ceramide

维持膜内正常状态:中性鞘磷脂酶 2 作为质膜神经酰胺的主要调节因子

Quadri, Zainuddin; Bieberich, Erhard

Editorial: The role of glial cells in the pathophysiology and treatment of Alzheimer's disease

社论:神经胶质细胞在阿尔茨海默病病理生理学和治疗中的作用

Crivelli, Simone M; Quadri, Zainuddin; van Kruining, Daan; Martinez-Martinez, Pilar; Bieberich, Erhard; Chatton, Jean-Yves

Abnormal Regulation of Mitochondrial Sphingolipids during Aging and Alzheimer's Disease.

衰老和阿尔茨海默病过程中线粒体鞘脂的异常调节

Crivelli Simone M, Quadri Zainuddin, Elsherbini Ahmed, Vekaria Hemendra J, Sullivan Patrick G, Zhi Wenbo, Martinez-Martinez Pilar, Spassieva Stefka D, Bieberich Erhard

Editorial: Meeting new challenges in translationally relevant neurodegenerative disease research

社论:迎接与转化相关的神经退行性疾病研究的新挑战

Latimer, Caitlin Shannon; Quadri, Zainuddin; Cook, David G

Extracellular vesicles in pharmacology: Novel approaches in diagnostics and therapy

细胞外囊泡在药理学中的应用:诊断和治疗的新方法

Quadri, Zainuddin; Elsherbini, Ahmed; Bieberich, Erhard

CCL2 Overexpression in the Brain Promotes Glial Activation and Accelerates Tau Pathology in a Mouse Model of Tauopathy.

脑内 CCL2 过表达促进胶质细胞活化,加速 Tau 蛋白病小鼠模型中的 Tau 蛋白病理

Joly-Amado Aurelie, Hunter Jordan, Quadri Zainuddin, Zamudio Frank, Rocha-Rangel Patricia V, Chan Deanna, Kesarwani Anisha, Nash Kevin, Lee Daniel C, Morgan Dave, Gordon Marcia N, Selenica Maj-Linda B

Overexpression of human wtTDP-43 causes impairment in hippocampal plasticity and behavioral deficits in CAMKII-tTa transgenic mouse model

人源wtTDP-43的过表达会导致CAMKII-tTa转基因小鼠模型海马可塑性受损和行为缺陷。

Quadri, Zainuddin; Johnson, Nicholas; Zamudio, Frank; Miller, Abraian; Peters, Melinda; Smeltzer, Shayna; Hunt, Jerry B Jr; Housley, Steven B; Brown, Breanna; Kraner, Susan; Norris, Christopher M; Nash, Kevin; Weeber, Edwin; Lee, Daniel C; Selenica, Maj-Linda B

TDP-43 loss of cellular function through aggregation requires additional structural determinants beyond its C-terminal Q/N prion-like domain.

TDP-43 通过聚集丧失细胞功能需要除其 C 端 Q/N 朊病毒样结构域之外的其他结构决定因素

Budini Mauricio, Romano Valentina, Quadri Zainuddin, Buratti Emanuele, Baralle Francisco E