Rimpy Dhingra相关文献与解析，生知库 | 链接生命科学的每一步

Mitochondrial autophagy and cell survival is regulated by the circadian Clock gene in cardiac myocytes during ischemic stress

缺血应激期间心肌细胞的线粒体自噬和细胞存活受昼夜节律基因调控

doi:10.1080/15548627.2021.1938913

Inna Rabinovich-Nikitin, Mina Rasouli, Cristine J Reitz, Illana Posen, Victoria Margulets, Rimpy Dhingra, Tarak N Khatua, James A Thliveris, Tami A Martino, Lorrie A Kirshenbaum

Impaired NF-κB signalling underlies cyclophilin D-mediated mitochondrial permeability transition pore opening in doxorubicin cardiomyopathy

NF-κB 信号传导受损是阿霉素心肌病中环丝氨酸蛋白酶 D 介导的线粒体通透性转换孔开放的基础

期刊:Cardiovascular Research

影响因子:10.2

doi:10.1093/cvr/cvz240

Rimpy Dhingra, Matthew Guberman, Inna Rabinovich-Nikitin, Jonathon Gerstein, Victoria Margulets, Hongying Gang, Nicholas Madden, James Thliveris, Lorrie A Kirshenbaum

Ageing-associated increase in SGLT2 disrupts mitochondrial/sarcoplasmic reticulum Ca2+ homeostasis and promotes cardiac dysfunction

与衰老相关的 SGLT2 增加会破坏线粒体/肌浆网 Ca2+ 稳态并导致心脏功能障碍

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.3

doi:10.1111/jcmm.15483

Yusuf Olgar, Erkan Tuncay, Sinan Degirmenci, Deniz Billur, Rimpy Dhingra, Lorrie Kirshenbaum, Belma Turan

Bnip3 mediates doxorubicin-induced cardiac myocyte necrosis and mortality through changes in mitochondrial signaling

Bnip3 通过改变线粒体信号介导阿霉素诱导的心肌细胞坏死和死亡

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1414665111

Rimpy Dhingra, Victoria Margulets, Subir Roy Chowdhury, James Thliveris, Davinder Jassal, Paul Fernyhough, Gerald W Dorn 2nd, Lorrie A Kirshenbaum

Bidirectional regulation of nuclear factor-κB and mammalian target of rapamycin signaling functionally links Bnip3 gene repression and cell survival of ventricular myocytes

核因子 κB 和哺乳动物雷帕霉素靶信号的双向调节在功能上将 Bnip3 基因抑制与心室肌细胞的细胞存活联系起来

期刊:Circulation-Heart Failure

影响因子:7.8

doi:10.1161/CIRCHEARTFAILURE.112.000061

Rimpy Dhingra, Hongying Gang, Yan Wang, Agnieszka K Biala, Yaron Aviv, Victoria Margulets, Andrew Tee, Lorrie A Kirshenbaum

NF-κB p65 Attenuates Cardiomyocyte PGC-1α Expression in Hypoxia

NF-κB p65 抑制缺氧条件下心肌细胞 PGC-1α 的表达

Mitochondrial autophagy and cell survival is regulated by the circadian Clock gene in cardiac myocytes during ischemic stress

缺血应激期间心肌细胞的线粒体自噬和细胞存活受昼夜节律基因调控

Impaired NF-κB signalling underlies cyclophilin D-mediated mitochondrial permeability transition pore opening in doxorubicin cardiomyopathy

NF-κB 信号传导受损是阿霉素心肌病中环丝氨酸蛋白酶 D 介导的线粒体通透性转换孔开放的基础

Ageing-associated increase in SGLT2 disrupts mitochondrial/sarcoplasmic reticulum Ca2+ homeostasis and promotes cardiac dysfunction

与衰老相关的 SGLT2 增加会破坏线粒体/肌浆网 Ca2+ 稳态并导致心脏功能障碍

Bnip3 mediates doxorubicin-induced cardiac myocyte necrosis and mortality through changes in mitochondrial signaling

Bnip3 通过改变线粒体信号介导阿霉素诱导的心肌细胞坏死和死亡

Bidirectional regulation of nuclear factor-κB and mammalian target of rapamycin signaling functionally links Bnip3 gene repression and cell survival of ventricular myocytes

核因子 κB 和哺乳动物雷帕霉素靶信号的双向调节在功能上将 Bnip3 基因抑制与心室肌细胞的细胞存活联系起来