Altered trafficking of Kv1-Kvβ2 leads to neuronal hyperexcitability and memory deficits in amyloid-β pathology.
Kv1-Kvβ2 转运异常导致淀粉样蛋白β病理中的神经元过度兴奋和记忆缺陷。
期刊:Molecular Neurodegeneration
影响因子:17.5
doi:10.1186/s13024-026-00936-2
Cho Young-Sin, Kim Seo-Hyun, Ryu Shin-Hyeon, Chung Chaelin, Lee Nahyun, Kim Namhoon, Jeong Muhah, Kim Youngwon, Gwak Jimin, Choi Se-Young, Saido Takaomi, Jung Yong-Keun