日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Constitutive, mosaic expression of TIE2 p.L914F during mouse development causes formation of venous malformation

小鼠发育过程中TIE2 p.L914F的组成型嵌合表达会导致静脉畸形的形成。

Bischoff, Lindsay J; Sherpa, Chhiring; Schrenk, Sandra; Boscolo, Elisa

Expression of mutant TIE2 p.L914F during mouse development causes embryonic lethality and defects in vascular remodeling

小鼠发育过程中突变型TIE2 p.L914F的表达会导致胚胎致死和血管重塑缺陷。

Bischoff, Lindsay J; Schrenk, Sandra; Soroko, Kara; Sherpa, Chhiring; Arasu, Ashok; Reynaud, Damien; Boscolo, Elisa

Hypoxia regulate developmental coronary angiogenesis potentially through VEGF-R2- and SOX17-mediated signaling.

缺氧可能通过 VEGF-R2 和 SOX17 介导的信号传导来调节发育性冠状动脉血管生成

Vitali Halie E, Kuschel Bryce, Sherpa Chhiring, Jones Brendan W, Jacob Nisha, Madiha Syeda A, Elliott Sam, Dziennik Eddie, Kreun Lily, Conatser Cora, Bhetwal Bhupal P, Sharma Bikram

Semaphorin 3A and 3F overexpression in TIE2 hyperactive endothelial cells contribute to the pathological lumen expansion in venous malformation.

TIE2 过度活跃的内皮细胞中信号素 3A 和 3F 的过度表达导致静脉畸形中病理性管腔扩张

Schrenk Sandra, Sherpa Chhiring, Bischoff Lindsay J, Cai Yuqi, Boscolo Elisa