Smeyne相关文献与解析，生知库 | 链接生命科学的每一步

Exposure to manganese during juvenile development increases microglial activation in the hippocampus following systemic infection with A/California/04/2009 Influenza A H1N1 virus

幼年发育期间接触锰会增加全身感染A/California/04/2009甲型H1N1流感病毒后海马体中小胶质细胞的活化。

期刊:Frontiers in Toxicology

影响因子:4.6

doi:10.3389/ftox.2026.1789730

Hager, Megan R; Schuller, Adam J; Yanouri, Omar A; Bantle, Collin M; Smeyne, Richard J; Tjalkens, Ronald B

Environmental exposures and familial background alter the induction of neuropathology and inflammation after SARS-CoV-2 infection.

环境因素和家族背景会改变 SARS-CoV-2 感染后神经病理学和炎症的诱发

期刊:Npj Parkinsons Disease

影响因子:8.2

doi:10.1038/s41531-025-00925-0

Chatterjee Debotri, Kurup Drishya, Smeyne Richard Jay

Templating of monomeric alpha-synuclein results in inflammation and SNpc dopamine neuron death in a genetic mouse model of induced synucleinopathy.

在诱导突触核蛋白病的基因小鼠模型中，单体α-突触核蛋白的模板化会导致炎症和黑质致密部多巴胺神经元死亡

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-025-10705-8

Byrne Matthew D, Petramfar Peyman, Lee Jae-Kyung, Smeyne Richard Jay

GUCY2C signaling limits dopaminergic neuron vulnerability to toxic insults

GUCY2C信号传导限制多巴胺能神经元对毒性损伤的易感性

期刊:Npj Parkinsons Disease

影响因子:8.2

doi:10.1038/s41531-024-00697-z

Cheslow, Lara; Byrne, Matthew; Kopenhaver, Jessica S; Iacovitti, Lorraine; Smeyne, Richard J; Snook, Adam E; Waldman, Scott A

Templating of Monomeric Alpha-Synuclein Induces Inflammation and SNpc Dopamine Neuron Death in a Genetic Mouse Model of Synucleinopathy

单体 α-突触核蛋白的模板化在突触核蛋白病的遗传小鼠模型中诱发炎症和 SNpc 多巴胺神经元死亡

期刊:

影响因子:

doi:10.21203/rs.3.rs-5269499/v1

Matthew D Byrne, Peyman Petramfar, Jae-Kyung Lee, Richard Jay Smeyne

神经

Immunology/Inflammation

发表日期：2024

文献求助收藏

Environmental exposures and familial background alter the induction of neuropathology and inflammation after SARS-CoV-2 infection

环境暴露和家族背景会改变SARS-CoV-2感染后神经病理学和炎症的诱发。

期刊:

影响因子:

doi:10.1101/2024.12.02.626375

Chatterjee, Debotri; Kurup, Drishya; Smeyne, Richard Jay

In memorium, Michael Zigmond, PhD

谨此缅怀迈克尔·齐格蒙德博士

期刊:Npj Parkinsons Disease

影响因子:8.2

doi:10.1038/s41531-024-00709-y

Santos-Lobato, Bruno Lopes; Smeyne, Richard J

发表日期：2023

文献求助收藏

Microglia-specific knock-out of NF-κB/IKK2 increases the accumulation of misfolded α-synuclein through the inhibition of p62/sequestosome-1-dependent autophagy in the rotenone model of Parkinson's disease

在帕金森病鱼藤酮模型中，小胶质细胞特异性敲除 NF-κB/IKK2 可通过抑制 p62/sequestosome-1 依赖性自噬增加错误折叠的 α-突触核蛋白的积累

期刊:Glia

影响因子:5.4

doi:10.1002/glia.24385

Savannah M Rocha, Kelly S Kirkley, Debotri Chatterjee, Tawfik A Aboellail, Richard J Smeyne, Ronald B Tjalkens

Neuroanatomical and neurochemical effects of prolonged social isolation in adult mice

成年小鼠长期社会隔离的神经解剖学和神经化学效应

期刊:Frontiers in Neuroanatomy

影响因子:2.3

doi:10.3389/fnana.2023.1190291

Heng, Vibol; Zigmond, Michael; Smeyne, Richard Jay

发表日期：2023

文献求助收藏

Persistent microglial activation following neonatal CMV infection mediates neurodegeneration.

新生儿巨细胞病毒感染后持续的小胶质细胞活化介导神经退行性变。

Exposure to manganese during juvenile development increases microglial activation in the hippocampus following systemic infection with A/California/04/2009 Influenza A H1N1 virus

幼年发育期间接触锰会增加全身感染A/California/04/2009甲型H1N1流感病毒后海马体中小胶质细胞的活化。

Environmental exposures and familial background alter the induction of neuropathology and inflammation after SARS-CoV-2 infection.

环境因素和家族背景会改变 SARS-CoV-2 感染后神经病理学和炎症的诱发

Templating of monomeric alpha-synuclein results in inflammation and SNpc dopamine neuron death in a genetic mouse model of induced synucleinopathy.

在诱导突触核蛋白病的基因小鼠模型中，单体α-突触核蛋白的模板化会导致炎症和黑质致密部多巴胺神经元死亡

GUCY2C signaling limits dopaminergic neuron vulnerability to toxic insults

GUCY2C信号传导限制多巴胺能神经元对毒性损伤的易感性

Templating of Monomeric Alpha-Synuclein Induces Inflammation and SNpc Dopamine Neuron Death in a Genetic Mouse Model of Synucleinopathy

单体 α-突触核蛋白的模板化在突触核蛋白病的遗传小鼠模型中诱发炎症和 SNpc 多巴胺神经元死亡

Environmental exposures and familial background alter the induction of neuropathology and inflammation after SARS-CoV-2 infection

环境暴露和家族背景会改变SARS-CoV-2感染后神经病理学和炎症的诱发。

In memorium, Michael Zigmond, PhD

谨此缅怀迈克尔·齐格蒙德博士

Microglia-specific knock-out of NF-κB/IKK2 increases the accumulation of misfolded α-synuclein through the inhibition of p62/sequestosome-1-dependent autophagy in the rotenone model of Parkinson's disease

在帕金森病鱼藤酮模型中，小胶质细胞特异性敲除 NF-κB/IKK2 可通过抑制 p62/sequestosome-1 依赖性自噬增加错误折叠的 α-突触核蛋白的积累

Neuroanatomical and neurochemical effects of prolonged social isolation in adult mice

成年小鼠长期社会隔离的神经解剖学和神经化学效应