日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The Role and Phagocytic Mechanisms of Alveolar Macrophages in Inflammatory Responses Induced by Diesel Exhaust Particles.

肺泡巨噬细胞在柴油机尾气颗粒诱发的炎症反应中的作用和吞噬机制。

Han Dongyang, Peng Renzhen, Pan Bin, Xie Yuanting, Shao Wenpu, Fang Xinyi, Li Jingyu, Hong Xiaoqing, Tu Wenyue, Muller-Deile Janina, Zhu Yaning, Li Weihua, Kan Haidong, Ying Zhekang, Xu Yanyi

Correction: Central IKKβ Inhibition prevents air pollution mediated peripheral inflammation and exaggeration of type II diabetes

更正:中枢 IKKβ 抑制可预防空气污染介导的外周炎症和 II 型糖尿病的加重

Liu, Cuiqing; Fonken, Laura K; Wang, Aixia; Maiseyeu, Andrei; Bai, Yuntao; Wang, Tse-Yao; Maurya, Santosh; Ko, Yi-An; Periasamy, Muthu; Dvonch, Timothy; Morishita, Masako; Brook, Robert D; Harkema, Jack; Ying, Zhekang; Mukherjee, Bhramar; Sun, Qinghua; Nelson, Randy J; Rajagopalan, Sanjay

Gut microbiota mediates ambient PM(2.5) exposure-induced abnormal glucose metabolism via short-chain fatty acids

肠道菌群通过短链脂肪酸介导环境PM(2.5)暴露引起的异常葡萄糖代谢

Shao, Wenpu; Pan, Bin; Li, Zhouzhou; Peng, Renzhen; Yang, Wenhui; Xie, Yuanting; Han, Dongyang; Fang, Xinyi; Li, Jingyu; Zhu, Yaning; Zhao, Zhuohui; Kan, Haidong; Ying, Zhekang; Xu, Yanyi

Timing determines programming of energy homeostasis by maternal PM(2.5) exposure in mouse models

在小鼠模型中,母体PM2.5暴露的时间决定了能量稳态的程序化调控

Wang, Shan; Peng, Renzhen; Chen, Hongxia; Han, Dongyang; Wu, Junfang; Xu, Yanyi; Ying, Zhekang

Deficiency of interleukin-6 receptor ameliorates PM(2.5) exposure-induced pulmonary dysfunction and inflammation but not abnormalities in glucose homeostasis

白细胞介素-6受体缺乏可改善PM(2.5)暴露引起的肺功能障碍和炎症,但不能改善葡萄糖稳态异常。

Peng, Renzhen; Yang, Wenhui; Shao, Wenpu; Pan, Bin; Zhu, Yaning; Zhang, Yubin; Kan, Haidong; Xu, Yanyi; Ying, Zhekang

cis-4-[(18)F]fluoro-L-proline Molecular Imaging Experimental Liver Fibrosis

顺式-4-[(18)F]氟-L-脯氨酸分子影像实验肝纤维化

Cao, Qi; Lu, Xin; Azad, Babak Behnam; Pomper, Martin; Smith, Mark; He, Jiang; Pi, Liya; Ren, Bin; Ying, Zhekang; Sichani, Babak Saboury; Morris, Michael; Dilsizian, Vasken

Developmental programming of obesity by maternal exposure to concentrated ambient PM(2.5) is maternally transmitted into the third generation in a mouse model

在小鼠模型中,母体暴露于高浓度环境PM2.5导致的肥胖发育编程可通过母系遗传至第三代。

Xu, Yanyi; Wang, Wanjun; Chen, Minjie; Zhou, Ji; Huang, Xingke; Tao, Shimin; Pan, Bin; Li, Zhouzhou; Xie, Xiaoyun; Li, Weihua; Kan, Haidong; Ying, Zhekang

Metabolomics analysis of a mouse model for chronic exposure to ambient PM(2.5)

代谢组学分析小鼠慢性暴露于环境PM(2.5)模型

Xu, Yanyi; Wang, Wanjun; Zhou, Ji; Chen, Minjie; Huang, Xingke; Zhu, Yaning; Xie, Xiaoyun; Li, Weihua; Zhang, Yuhao; Kan, Haidong; Ying, Zhekang

Concentrated Ambient PM(2.5)-Induced Inflammation and Endothelial Dysfunction in a Murine Model of Neural IKK2 Deficiency

高浓度环境PM(2.5)诱导神经IKK2缺陷小鼠模型发生炎症和内皮功能障碍

Chen, Minjie; Qin, Xiaobo; Qiu, Lianglin; Chen, Sufang; Zhou, Huifen; Xu, Yanyi; Hu, Ziying; Zhang, Yuhao; Cao, Qi; Ying, Zhekang

Exposure to concentrated ambient PM(2.5) alters the composition of gut microbiota in a murine model

小鼠模型中,暴露于高浓度环境PM2.5会改变肠道菌群的组成。

Wang, Wanjun; Zhou, Ji; Chen, Minjie; Huang, Xingke; Xie, Xiaoyun; Li, Weihua; Cao, Qi; Kan, Haidong; Xu, Yanyi; Ying, Zhekang