详细信息
背景信息
Patented technology Our RabMAb ® technology is a patented hybridoma-based technology for making rabbit monoclonal antibodies. For details on our patents, please refer to RabMAb ® patents. What are the advantages of a recombinant monoclonal antibody? This product is a recombinant monoclonal antibody, which offers several advantages including - High batch-to-batch consistency and reproducibility - Improved sensitivity and specificity - Long-term security of supply - Animal-free batch production For more information, read more on recombinant antibodies.
文献引用 (60)
Neuroplasticity After Hypoxic-Ischemic Brain Injury in Neonatal Pigs Based on Time-Dependent Behavior of (1)H-MRS-Tau Protein and Synaptic Associated Proteins and Synaptic Structure Analysis.
基于（1）H-MRS-Tau蛋白和突触相关蛋白的时间依赖性行为和突触结构分析，研究新生猪缺氧缺血性脑损伤后的神经可塑性
Kv2/Kv6.4 Heteromeric Potassium Channels Are Expressed in Spinal Motoneurons and Localized at C-Bouton Synapses
Kv2/Kv6.4异源钾通道在脊髓运动神经元中表达，并定位于C-突触。
Increased Synapse Elimination by Inflammatory Cells Contributes to Long-lasting Post-Stroke Memory Dysfunction in Old Mice.
炎症细胞对突触的破坏增加，导致老年小鼠中风后长期记忆功能障碍
Induced neural progenitor cells and iPS-neurons from major depressive disorder patients show altered bioenergetics and electrophysiological properties
重度抑郁症患者的诱导神经祖细胞和 iPS 神经元表现出改变的生物能量学和电生理学特性
Normobaric hyperoxia alleviates complement C3-mediated synaptic pruning and brain injury after intracerebral hemorrhage
常压高氧减轻脑出血后补体C3介导的突触修剪和脑损伤
Annexin A6 mitigates neurological deficit in ischemia/reperfusion injury by promoting synaptic plasticity
膜联蛋白 A6 通过促进突触可塑性减轻缺血/再灌注损伤造成的神经功能缺损
Effects of early tooth loss on chronic stress and progression of neuropathogenesis of Alzheimer's disease in adult Alzheimer's model AppNL-G-F mice
早期牙齿脱落对成年阿尔茨海默病模型 AppNL-GF 小鼠慢性应激及神经病理学进展的影响
A Comprehensive Functional Investigation of the Human Translocator Protein 18 kDa (TSPO) in a Novel Human Neuronal Cell Knockout Model
在新型人类神经元细胞敲除模型中对人类转运蛋白 18 kDa (TSPO) 进行全面的功能研究
Effects on Synaptic Plasticity Markers in Fetal Mice and HT22 Neurons upon F-53B Exposure: The Role of PKA Cytoplasmic Retention
F-53B 暴露对胎儿小鼠和 HT22 神经元突触可塑性标志物的影响：PKA 细胞质保留的作用
- 1
- 2
- 3
- 4
- 5
- 6