详细信息
背景信息
What is this antibody validated in? Anti-Twist antibody [10E4E6] (ab175430) is a mouse monoclonal antibody and is validated for use in Western Blot (WB), Flow Cytometry (Flow Cyt), Immunohistochemistry (IHC-P), Immunocytochemistry/immunofluorescence (ICC/IF) in Human, Mouse, samples. What is the molecular weight of Twist? Anti-Twist [10E4E6] (ab175430) specifically detects a band for Twist (UniProt: Q15672) at a molecular weight of 21kDa. Trusted by the scientific community Anti-Twist [10E4E6] (ab175430) was first used in a scientific publication in 2013 and has been cited over 80 times in peer-reviewed journals.
文献引用 (98)
TWIST1 drives endothelial-to-mesenchymal-transition to stabilize atherosclerotic plaques.
TWIST1 驱动内皮细胞向间质细胞转化，从而稳定动脉粥样硬化斑块。
Overexpression of EMT-related transcription factors SNAI1 and ZEB1 is associated with more aggressive clinicopathological features of pancreatic cancer.
EMT相关转录因子SNAI1和ZEB1的过度表达与胰腺癌更具侵袭性的临床病理特征相关。
USP9X integrates TGF-β and hypoxia signalings to promote ovarian cancer chemoresistance via HIF-2α-maintained stemness.
USP9X整合TGF-β和缺氧信号，通过HIF-2α维持干性来促进卵巢癌的化疗耐药性
Knockdown of eIF3a alleviates pulmonary arterial hypertension by inhibiting endothelial-to-mesenchymal transition via TGFβ1/SMAD pathway.
敲除 eIF3a 可通过 TGFβ1/SMAD 通路抑制内皮细胞向间质细胞的转化，从而缓解肺动脉高压
FAM64A contributes to ovarian cancer proliferation and metastasis by suppressing TWIST1 ubiquitination and degradation.
FAM64A 通过抑制 TWIST1 泛素化和降解，促进卵巢癌的增殖和转移
Dose-dependent induction of epithelial-mesenchymal transition in 3D melanoma models by non-thermal plasma treatment.
非热等离子体治疗可剂量依赖性地诱导 3D 黑色素瘤模型中的上皮-间质转化。
Knockdown of CCNB2 inhibits the tumorigenesis of gastric cancer by regulation of the PI3K/Akt pathway
敲低CCNB2通过调控PI3K/Akt通路抑制胃癌发生
S100a4 promotes metastatic transformation in non-metastatic liver cancer cells through NMIIa binding: mechanistic insights.
S100a4 通过 NMIIa 结合促进非转移性肝癌细胞的转移转化：机制研究
Jumonji domain-containing protein 6 promotes gastric cancer progression: Modulating immune evasion through autophagy and oxidative stress pathways
含 Jumonji 结构域的蛋白 6 促进胃癌进展：通过自噬和氧化应激途径调节免疫逃避
Cuproptosis-Related Gene FDX1 Suppresses the Growth and Progression of Colorectal Cancer by Retarding EMT Progress
杯状凋亡相关基因FDX1通过延缓EMT进程抑制结直肠癌的生长和进展
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